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V R Sutton

Showing results (41-50 of 53) with videos related to

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The Journal of Experimental Medicine|November 21, 2000
Initiation of apoptosis by granzyme B requires direct cleavage of bid, but not direct granzyme B-mediated caspase activationV R Sutton, J E Davis, M Cancilla, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 6, 2001
The histone deacetylase inhibitor and chemotherapeutic agent suberoylanilide hydroxamic acid (SAHA) induces a cell-death pathway characterized by cleavage of Bid and production of reactive oxygen speciesA A Ruefli, M J Ausserlechner, D Bernhard, et al.
American Journal of Medical Genetics|June 24, 1998
Delineation of the common critical region in Williams syndrome and clinical correlation of growth, heart defects, ethnicity, and parental originY Q Wu, V R Sutton, E Nickerson, et al.
The British Journal of Dermatology|June 14, 2005
Lack of apoptosis of Sézary cells in the circulation following oral bexarotene therapyS Brennand, V R Sutton, J Biagi, et al.
American Journal of Human Genetics|October 27, 1997
Chromosome 1p36 deletions: the clinical phenotype and molecular characterization of a common newly delineated syndromeS K Shapira, C McCaskill, H Northrup, et al.
Human Mutation|June 20, 2003
Epigenetic detection of human chromosome 14 uniparental disomyS K Murphy, A A Wylie, K J Coveler, et al.
Cell Death & Disease|July 6, 2012
Granzyme B triggers a prolonged pressure to die in Bcl-2 overexpressing cells, defining a window of opportunity for effective treatment with ABT-737V R Sutton, K Sedelies, G Dewson, et al.
Cell Death and Differentiation|September 17, 2005
Functional dissociation of DeltaPsim and cytochrome c release defines the contribution of mitochondria upstream of caspase activation during granzyme B-induced apoptosisN J Waterhouse, K A Sedelies, V R Sutton, et al.
Cell Death and Differentiation|January 19, 2008
Blocking granule-mediated death by primary human NK cells requires both protection of mitochondria and inhibition of caspase activityK A Sedelies, A Ciccone, C J P Clarke, et al.
Osteoporosis International : a Journal Established As Result of Cooperation Between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA|February 28, 2013
Next-generation sequencing for disorders of low and high bone mineral densityG Sule, P M Campeau, V W Zhang, et al.
Pageof 6

Showing results (41-50 of 53) with videos related to

Sort By:
Pageof 6
The Journal of Experimental Medicine|November 21, 2000
Initiation of apoptosis by granzyme B requires direct cleavage of bid, but not direct granzyme B-mediated caspase activationV R Sutton, J E Davis, M Cancilla, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 6, 2001
The histone deacetylase inhibitor and chemotherapeutic agent suberoylanilide hydroxamic acid (SAHA) induces a cell-death pathway characterized by cleavage of Bid and production of reactive oxygen speciesA A Ruefli, M J Ausserlechner, D Bernhard, et al.
American Journal of Medical Genetics|June 24, 1998
Delineation of the common critical region in Williams syndrome and clinical correlation of growth, heart defects, ethnicity, and parental originY Q Wu, V R Sutton, E Nickerson, et al.
The British Journal of Dermatology|June 14, 2005
Lack of apoptosis of Sézary cells in the circulation following oral bexarotene therapyS Brennand, V R Sutton, J Biagi, et al.
American Journal of Human Genetics|October 27, 1997
Chromosome 1p36 deletions: the clinical phenotype and molecular characterization of a common newly delineated syndromeS K Shapira, C McCaskill, H Northrup, et al.
Human Mutation|June 20, 2003
Epigenetic detection of human chromosome 14 uniparental disomyS K Murphy, A A Wylie, K J Coveler, et al.
Cell Death & Disease|July 6, 2012
Granzyme B triggers a prolonged pressure to die in Bcl-2 overexpressing cells, defining a window of opportunity for effective treatment with ABT-737V R Sutton, K Sedelies, G Dewson, et al.
Cell Death and Differentiation|September 17, 2005
Functional dissociation of DeltaPsim and cytochrome c release defines the contribution of mitochondria upstream of caspase activation during granzyme B-induced apoptosisN J Waterhouse, K A Sedelies, V R Sutton, et al.
Cell Death and Differentiation|January 19, 2008
Blocking granule-mediated death by primary human NK cells requires both protection of mitochondria and inhibition of caspase activityK A Sedelies, A Ciccone, C J P Clarke, et al.
Osteoporosis International : a Journal Established As Result of Cooperation Between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA|February 28, 2013
Next-generation sequencing for disorders of low and high bone mineral densityG Sule, P M Campeau, V W Zhang, et al.
Pageof 6