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Clinical and Experimental Immunology|January 27, 2015
Complement activation and expression during chronic relapsing experimental autoimmune encephalomyelitis in the Biozzi ABH mouseV Ramaglia, S J Jackson, T R Hughes, et al.
Neuroscience Letters|November 17, 2012
Deletion of Crry, the murine ortholog of the sporadic Alzheimer's disease risk gene CR1, impacts tau phosphorylation and brain CFHR Killick, T R Hughes, B P Morgan, et al.
Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration|September 1, 1995
A functional role for corpora amylacea based on evidence from complement studiesS K Singhrao, B P Morgan, J W Neal, et al.
British Journal of Pharmacology|September 25, 2007
Cannabinoid control of neuroinflammation related to multiple sclerosisD Baker, S J Jackson, G Pryce
Clinical and Experimental Immunology|November 17, 2004
Blockade of the C5a receptor fails to protect against experimental autoimmune encephalomyelitis in ratsB P Morgan, M Griffiths, H Khanom, et al.
The American Journal of Pathology|January 1, 1997
Expression of the receptor for complement C5a (CD88) is up-regulated on reactive astrocytes, microglia, and endothelial cells in the inflamed human central nervous systemP Gasque, S K Singhrao, J W Neal, et al.
Immunology|November 1, 1993
Immunolocalization and characterization of the rat analogue of human CD59 in kidney and glomerular cellsT R Hughes, S Meri, M Davies, et al.
Clinical and Experimental Immunology|October 13, 2006
Complement membrane attack is required for endplate damage and clinical disease in passive experimental myasthenia gravis in Lewis ratsJ Chamberlain-Banoub, J W Neal, M Mizuno, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|October 26, 1999
Differential expression of individual complement regulators in the brain and choroid plexusS K Singhrao, J W Neal, N K Rushmere, et al.
The American Journal of Pathology|September 12, 2000
Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysisS K Singhrao, J W Neal, N K Rushmere, et al.
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