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The American Journal of Physiology|December 1, 1985
Phase resetting and annihilation in a mathematical model of sinus nodeV S Reiner, C AntzelevitchJournal of Electrocardiology|January 10, 2002
Molecular biology and cellular mechanisms of Brugada and long QT syndromes in infants and young childrenC AntzelevitchCardiology Clinics|February 1, 1983
Clinical application of new concepts of parasystole, reflection, and tachycardiaC AntzelevitchCurrent Opinion in Cardiology|December 22, 2000
Basic mechanisms of reentrant arrhythmiasC AntzelevitchThe Journal of Physiology|May 15, 2001
Molecular basis for the transmural distribution of the transient outward currentC AntzelevitchCurrent Opinion in Cardiology|June 8, 1999
Ion channels and ventricular arrhythmias: cellular and ionic mechanisms underlying the Brugada syndromeC AntzelevitchJournal of the American College of Cardiology|March 15, 2000
Differential effects of beta-adrenergic agonists and antagonists in LQT1, LQT2 and LQT3 models of the long QT syndromeW Shimizu, C AntzelevitchJournal of Electrocardiology|February 25, 2000
Cellular basis for long QT, transmural dispersion of repolarization, and torsade de pointes in the long QT syndromeW Shimizu, C AntzelevitchJournal of Cardiovascular Electrophysiology|May 16, 2000
Block of I(Ks) does not induce early afterdepolarization activity but promotes beta-adrenergic agonist-induced delayed afterdepolarization activityA Burashnikov, C AntzelevitchCirculation|March 23, 1999
Cellular and ionic basis for T-wave alternans under long-QT conditionsW Shimizu, C AntzelevitchPageof 9