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Oncogene|July 6, 2001
Low concentrations of paclitaxel induce cell type-dependent p53, p21 and G1/G2 arrest instead of mitotic arrest: molecular determinants of paclitaxel-induced cytotoxicityP Giannakakou, R Robey, T Fojo, et al.The Lancet. Respiratory Medicine|December 20, 2016
Expected effects of adopting a 9 month regimen for multidrug-resistant tuberculosis: a population modelling analysisEmily A Kendall, Anthony T Fojo, David W DowdyGan to Kagaku Ryoho. Cancer & Chemotherapy|January 9, 1998
[Expression of DNA topoisomerases (I, II alpha, II beta) mRNA in etoposide- and mAMSA-resistant cell lines]Y Matsumoto, H Takano, S Nagao, et al.Annals of Oncology : Official Journal of the European Society for Medical Oncology|April 29, 2017
Do patient access schemes for high-cost cancer drugs deliver value to society?-lessons from the NHS Cancer Drugs FundA Aggarwal, T Fojo, C Chamberlain, et al.Leukemia|July 10, 1999
Mitogen-activated protein kinase pathway is dispensable for microtubule-active drug-induced Raf-1/Bcl-2 phosphorylation and apoptosis in leukemia cellsM V Blagosklonny, Y Chuman, R C Bergan, et al.Japanese Journal of Cancer Research : Gann|July 28, 2001
Expression of drug resistance genes in VP-16 and mAMSA-selected human carcinoma cellsY Matsumoto, H Takano, K Kunishio, et al.International Journal of Cancer|January 14, 1998
Combinations of paclitaxel and vinblastine and their effects on tubulin polymerization and cellular cytotoxicity: characterization of a synergistic scheduleP Giannakakou, L Villalba, H Li, et al.Japanese Journal of Cancer Research : Gann|October 26, 2001
Incidence of mutation and deletion in topoisomerase II alpha mRNA of etoposide and mAMSA-resistant cell linesY Matsumoto, H Takano, K Kunishio, et al.Japanese Journal of Cancer Research : Gann|July 28, 2001
Hypophosphorylation of topoisomerase IIalpha in etoposide (VP-16)-resistant human carcinoma cell lines associated with carboxy-terminal truncationY Matsumoto, H Takano, K Kunishio, et al.Experimental Cell Research|October 15, 1998
Inhibitors of transcription, proteasome inhibitors, and DNA-damaging drugs differentially affect feedback of p53 degradationW G An, Y Chuman, T Fojo, et al.Pageof 24