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Mayo Clinic Proceedings|November 1, 1984
Passive transfer of arthritis to mice by injection of human anti-type II collagen antibodyP H Wooley, H S Luthra, S K Singh, et al.
Immunogenetics|October 29, 1999
H2-E transgenic class II-negative mice can distinguish self from nonself in susceptibility to heterologous thyroglobulins in autoimmune thyroiditisQ Wan, R Shah, D J McCormick, et al.
The Journal of Experimental Medicine|January 1, 1996
HLA-DQ8 transgenic mice are highly susceptible to collagen-induced arthritis: a novel model for human polyarthritisG H Nabozny, J M Baisch, S Cheng, et al.
Human Immunology|July 30, 1999
NOD background genes influence T cell responses to GAD 65 in HLA-DQ8 transgenic miceR S Abraham, S B Wilson, N F de Souza, et al.
Human Immunology|February 23, 1999
Peptide binding alpha1alpha2 domain of HLA-B27 contributes to the disease pathogenesis in transgenic miceS D Khare, S Lee, M J Bull, et al.
The Journal of Clinical Investigation|December 31, 1997
HLA-DQB1 polymorphism determines incidence, onset, and severity of collagen-induced arthritis in transgenic mice. Implications in human rheumatoid arthritisD S Bradley, G H Nabozny, S Cheng, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 15, 1995
Primary hormonogenic sites as conserved autoepitopes on thyroglobulin in murine autoimmune thyroiditis. Secondary role of iodinationY C Kong, D J McCormick, Q Wan, et al.
Human Immunology|October 16, 2001
HLA-DQ8 transgenic and NOD mice recognize different epitopes within the cytoplasmic region of the tyrosine phosphatase-like molecule, IA-2Y C Kudva, Y J Deng, R Govindarajan, et al.
The Journal of Clinical Investigation|June 8, 2000
Hla-DR2-restricted responses to proteolipid protein 95-116 peptide cause autoimmune encephalitis in transgenic miceK Kawamura, T Yamamura, K Yokoyama, et al.
Clinical Immunology and Immunopathology|November 5, 1997
Primary hormonogenic sites as conserved autoepitopes on thyroglobulin in murine autoimmune thyroiditis: role of MHC class IIQ Wan, R W Motte, D J McCormick, et al.
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