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Showing results (31-40 of 39) with videos related to

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Annals of Neurology|October 3, 2009
Early-onset absence epilepsy caused by mutations in the glucose transporter GLUT1Arvid Suls, Saul A Mullen, Yvonne G Weber, et al.
Molecular Neurodegeneration|August 12, 2021
Microglial inclusions and neurofilament light chain release follow neuronal α-synuclein lesions in long-term brain slice culturesMelanie Barth, Mehtap Bacioglu, Niklas Schwarz, et al.
The Journal of Clinical Investigation|September 21, 2021
Hyperexcitable interneurons trigger cortical spreading depression in an Scn1a migraine modelEva Auffenberg, Ulrike Bs Hedrich, Raffaella Barbieri, et al.
Journal of Neurophysiology|February 27, 2025
Comprehensive analysis of human dendritic spine morphology and densityKerstin D Schünemann, Roxanne M Hattingh, Matthijs B Verhoog, et al.
The Journal of Clinical Investigation|May 3, 2008
GLUT1 mutations are a cause of paroxysmal exertion-induced dyskinesias and induce hemolytic anemia by a cation leakYvonne G Weber, Alexander Storch, Thomas V Wuttke, et al.
Brain : a Journal of Neurology|June 26, 2008
Paroxysmal exercise-induced dyskinesia and epilepsy is due to mutations in SLC2A1, encoding the glucose transporter GLUT1Arvid Suls, Peter Dedeken, Karolien Goffin, et al.
Brain : a Journal of Neurology|June 5, 2025
The genetic and phenotypic spectrum of GABRB1-related disordersCharissa Millevert, Anthony Sze Hon Kan, Moritz Hanke, et al.
Neurology|January 6, 2017
Mutations in GABRB3: From febrile seizures to epileptic encephalopathiesRikke S Møller, Thomas V Wuttke, Ingo Helbig, et al.
Nature Neuroscience|August 26, 2020
A community-based transcriptomics classification and nomenclature of neocortical cell typesRafael Yuste, Michael Hawrylycz, Nadia Aalling, et al.
Pageof 4

Showing results (31-40 of 39) with videos related to

Sort By:
Pageof 4
You have reached the last page of results.This site can display upto 39 results.
Annals of Neurology|October 3, 2009
Early-onset absence epilepsy caused by mutations in the glucose transporter GLUT1Arvid Suls, Saul A Mullen, Yvonne G Weber, et al.
Molecular Neurodegeneration|August 12, 2021
Microglial inclusions and neurofilament light chain release follow neuronal α-synuclein lesions in long-term brain slice culturesMelanie Barth, Mehtap Bacioglu, Niklas Schwarz, et al.
The Journal of Clinical Investigation|September 21, 2021
Hyperexcitable interneurons trigger cortical spreading depression in an Scn1a migraine modelEva Auffenberg, Ulrike Bs Hedrich, Raffaella Barbieri, et al.
Journal of Neurophysiology|February 27, 2025
Comprehensive analysis of human dendritic spine morphology and densityKerstin D Schünemann, Roxanne M Hattingh, Matthijs B Verhoog, et al.
The Journal of Clinical Investigation|May 3, 2008
GLUT1 mutations are a cause of paroxysmal exertion-induced dyskinesias and induce hemolytic anemia by a cation leakYvonne G Weber, Alexander Storch, Thomas V Wuttke, et al.
Brain : a Journal of Neurology|June 26, 2008
Paroxysmal exercise-induced dyskinesia and epilepsy is due to mutations in SLC2A1, encoding the glucose transporter GLUT1Arvid Suls, Peter Dedeken, Karolien Goffin, et al.
Brain : a Journal of Neurology|June 5, 2025
The genetic and phenotypic spectrum of GABRB1-related disordersCharissa Millevert, Anthony Sze Hon Kan, Moritz Hanke, et al.
Neurology|January 6, 2017
Mutations in GABRB3: From febrile seizures to epileptic encephalopathiesRikke S Møller, Thomas V Wuttke, Ingo Helbig, et al.
Nature Neuroscience|August 26, 2020
A community-based transcriptomics classification and nomenclature of neocortical cell typesRafael Yuste, Michael Hawrylycz, Nadia Aalling, et al.
Pageof 4