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Biochimica Et Biophysica Acta. Molecular Basis of Disease|July 26, 2017
Inhibition of mitochondrial fission prevents hypoxia-induced metabolic shift and cellular proliferation of pulmonary arterial smooth muscle cellsValentina Parra, Roberto Bravo-Sagua, Ignacio Norambuena-Soto, et al.
Free Radical Biology & Medicine|December 3, 2023
HERPUD1 governs tumor cell mitochondrial function via inositol 1,4,5-trisphosphate receptor-mediated calcium signalingFelipe Paredes, Mario Navarro-Marquez, Clara Quiroga, et al.
Frontiers in Endocrinology|May 29, 2019
Polycystin-2 Is Required for Starvation- and Rapamycin-Induced Atrophy in MyotubesCatalina Kretschmar, Daniel Peña-Oyarzun, Cecilia Hernando, et al.
International Review of Cell and Molecular Biology|January 16, 2013
Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integrationRoberto Bravo, Valentina Parra, Damián Gatica, et al.
Journal of Cell Science|April 30, 2014
Mitochondrial fission is required for cardiomyocyte hypertrophy mediated by a Ca2+-calcineurin signaling pathwayChristian Pennanen, Valentina Parra, Camila López-Crisosto, et al.
Frontiers in Cell and Developmental Biology|April 9, 2026
Transcriptional regulatory network analysis uncovers modular gene control and potential key regulators in diabetic cardiomyopathyFernanda Fredericksen, Víctor Aliaga-Tobar, Sebastián Aedo-Cares, et al.
Circulation Research|January 25, 2018
Down Syndrome Critical Region 1 Gene, <i>Rcan1</i>, Helps Maintain a More Fused Mitochondrial NetworkValentina Parra, Francisco Altamirano, Carolina P Hernández-Fuentes, et al.
Cell Cycle (Georgetown, Tex.)|June 5, 2014
Dexamethasone-induced autophagy mediates muscle atrophy through mitochondrial clearanceRodrigo Troncoso, Felipe Paredes, Valentina Parra, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|May 23, 2024
PKD2 regulates autophagy and forms a protein complex with BECN1 at the primary cilium of hypothalamic neuronal cellsCamila García-Navarrete, Catalina Kretschmar, Jorge Toledo, et al.
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