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Nature Medicine|January 2, 2007
Anorectic estrogen mimics leptin's effect on the rewiring of melanocortin cells and Stat3 signaling in obese animalsQian Gao, Gabor Mezei, Yongzhan Nie, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 10, 2007
Continuous fat oxidation in acetyl-CoA carboxylase 2 knockout mice increases total energy expenditure, reduces fat mass, and improves insulin sensitivityCheol Soo Choi, David B Savage, Lutfi Abu-Elheiga, et al.
Biorxiv : the Preprint Server for Biology|June 4, 2025
Metabolic reprogramming by endothelial ANGPTL4 depletion protects against diabetic kidney diseaseSwayam Prakash Srivastava, Han Zhou, Rachel Shenoi, et al.
Cell Reports|October 26, 2017
Absence of Carbohydrate Response Element Binding Protein in Adipocytes Causes Systemic Insulin Resistance and Impairs Glucose TransportArchana Vijayakumar, Pratik Aryal, Jennifer Wen, et al.
Cell Metabolism|February 6, 2007
Aging-associated reductions in AMP-activated protein kinase activity and mitochondrial biogenesisRichard M Reznick, Haihong Zong, Ji Li, et al.
Nature Medicine|May 8, 2012
Hepatic Hdac3 promotes gluconeogenesis by repressing lipid synthesis and sequestrationZheng Sun, Russell A Miller, Rajesh T Patel, et al.
Cell Reports|February 22, 2018
Genetic Ablation of miR-33 Increases Food Intake, Enhances Adipose Tissue Expansion, and Promotes Obesity and Insulin ResistanceNathan L Price, Abhishek K Singh, Noemi Rotllan, et al.
Nucleic Acids Research|November 17, 2014
The H19/let-7 double-negative feedback loop contributes to glucose metabolism in muscle cellsYuan Gao, Fuju Wu, Jichun Zhou, et al.
Science Advances|December 4, 2024
Renal Angptl4 is a key fibrogenic molecule in progressive diabetic kidney diseaseSwayam Prakash Srivastava, Han Zhou, Rachel Shenoi, et al.
The Journal of Clinical Investigation|June 16, 2007
Overexpression of uncoupling protein 3 in skeletal muscle protects against fat-induced insulin resistanceCheol Soo Choi, Jonathan J Fillmore, Jason K Kim, et al.
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