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Molecular and Cellular Neurosciences|February 17, 2015
Autophagy receptor defects and ALS-FTLDVeronika Majcher, Alice Goode, Victoria James, et al.Molecular and Cellular Neurosciences|August 25, 2016
ALS-FTLD associated mutations of SQSTM1 impact on Keap1-Nrf2 signallingAlice Goode, Sarah Rea, Melanie Sultana, et al.Expert Reviews in Molecular Medicine|October 2, 2007
The molecular pathogenesis of Paget disease of boneRobert LayfieldAutophagy|May 10, 2016
Defective recognition of LC3B by mutant SQSTM1/p62 implicates impairment of autophagy as a pathogenic mechanism in ALS-FTLDAlice Goode, Kevin Butler, Jed Long, et al.Experimental Cell Research|February 4, 2014
SQSTM1 mutations--bridging Paget disease of bone and ALS/FTLDSarah L Rea, Veronika Majcher, Mark S Searle, et al.BMC Biochemistry|December 6, 2007
Ubiquitin-mediated signalling and Paget's disease of boneRobert Layfield, Barry ShawProteomics|April 4, 2008
The 6(th) East Midlands Proteomics Workshop supported by the BSPR and BMSS 14 November 2007, Nottingham, UKRobert Layfield, Balwir Matharoo-BallBiochemical Society Transactions|May 17, 2008
Disruption of ubiquitin-mediated processes in diseases of the brain and boneRobert Layfield, Mark S SearleBone|November 3, 2012
The S349T mutation of SQSTM1 links Keap1/Nrf2 signalling to Paget's disease of boneTao Wright, Sarah L Rea, Alice Goode, et al.Clinical Medicine (London, England)|September 11, 2021
Images of the month 2: Pulmonary artery pseudoaneurysm formation within uterine leiomyosarcoma metastasesDivya Vaid, Veronika Majcher, Nicholas Heptonstall, et al.Pageof 16