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Proceedings of the National Academy of Sciences of the United States of America|August 1, 2006
The Wilms tumor gene, Wt1, is required for Sox9 expression and maintenance of tubular architecture in the developing testisFei Gao, Sourindra Maiti, Nargis Alam, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 17, 2015
Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablationLianjun Zhang, Min Chen, Qing Wen, et al.
The Journal of Clinical Investigation|December 3, 2010
Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylationQianghua Hu, Fei Gao, Weihua Tian, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 22, 2013
Rare variant detection using family-based sequencing analysisGang Peng, Yu Fan, Timothy B Palculict, et al.
Oncotarget|November 20, 2018
Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in miceColleen E Annesley, Cara Rabik, Amy S Duffield, et al.
Development (Cambridge, England)|November 27, 2016
Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expressionMin Chen, Lianjun Zhang, Xiuhong Cui, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|December 30, 2010
WT1 mutation and 11P15 loss of heterozygosity predict relapse in very low-risk wilms tumors treated with surgery alone: a children's oncology group studyElizabeth J Perlman, Paul E Grundy, James R Anderson, et al.
Stem Cell Reports|July 29, 2014
Wilms' tumor blastemal stem cells dedifferentiate to propagate the tumor bulkRachel Shukrun, Naomi Pode-Shakked, Oren Pleniceanu, et al.
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