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Proceedings of the National Academy of Sciences of the United States of America|January 16, 2026
Proteasomal proteolysis in p62 condensates directs tumor suppression or growth depending on their subcellular localizationChen Lulu-Shimron, Zhiwen Luo, Vera Brekhman, et al.
Endocrinology|October 20, 2011
Heparanase is highly expressed and regulates proliferation in GH-secreting pituitary tumor cellsHadara Rubinfeld, Victoria Cohen-Kaplan, Dvora Nass, et al.
The Journal of Biological Chemistry|June 26, 2010
Heparanase 2 interacts with heparan sulfate with high affinity and inhibits heparanase activityFlonia Levy-Adam, Sari Feld, Victoria Cohen-Kaplan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 17, 2009
A novel human heparanase splice variant, T5, endowed with protumorigenic characteristicsUri Barash, Victoria Cohen-Kaplan, Gil Arvatz, et al.
Molecular Cell|September 22, 2023
Regulation of nucleo-cytosolic 26S proteasome translocation by aromatic amino acids via mTOR is essential for cell survival under stressIdo Livneh, Victoria Cohen-Kaplan, Bertrand Fabre, et al.
Pacing and Clinical Electrophysiology : PACE|February 20, 2024
Ser194Leu DSG2 mutation, associated with arrhythmogenic left ventricular cardiomyopathy and ventricular tachycardiaMiry Blich, Yaniv Zohar, Victoria Cohen-Kaplan, et al.
Cell Death and Differentiation|September 12, 2024
Inhibition of nucleo-cytoplasmic proteasome translocation by the aromatic amino acids or silencing Sestrin3-their sensing mediator-is tumor suppressiveIdo Livneh, Bertrand Fabre, Gilad Goldhirsh, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|November 20, 2012
Macrophage activation by heparanase is mediated by TLR-2 and TLR-4 and associates with plaque progressionMiry Blich, Amnon Golan, Gil Arvatz, et al.
Cell|April 11, 2015
KPC1-mediated ubiquitination and proteasomal processing of NF-κB1 p105 to p50 restricts tumor growthYelena Kravtsova-Ivantsiv, Inna Shomer, Victoria Cohen-Kaplan, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 19, 2022
Cross-talk between mutant p53 and p62/SQSTM1 augments cancer cell migration by promoting the degradation of cell adhesion proteinsSaptaparna Mukherjee, Martino Maddalena, YiQing Lü, et al.
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