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Biorxiv : the Preprint Server for Biology|April 22, 2024
Postsynaptic BMP signaling regulates myonuclear properties in <i>Drosophila</i> larval musclesVictoria E von Saucken, Stefanie E Windner, Mary K Baylies
Molecular Neurodegeneration|August 4, 2017
TREM2 in Neurodegenerative DiseasesTaylor R Jay, Victoria E von Saucken, Gary E Landreth
Neurobiology of Disease|September 5, 2020
The effect of amyloid on microglia-neuron interactions before plaque onset occurs independently of TREM2 in a mouse model of Alzheimer's diseaseVictoria E von Saucken, Taylor R Jay, Gary E Landreth
The Journal of Cell Biology|October 30, 2024
Postsynaptic BMP signaling regulates myonuclear properties in Drosophila larval musclesVictoria E von Saucken, Stefanie E Windner, Giovanna Armetta, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 9, 2015
KTKEGV repeat motifs are key mediators of normal α-synuclein tetramerization: Their mutation causes excess monomers and neurotoxicityUlf Dettmer, Andrew J Newman, Victoria E von Saucken, et al.
Biorxiv : the Preprint Server for Biology|December 4, 2023
Muscle cofilin alters neuromuscular junction postsynaptic development to strengthen functional neurotransmissionBriana Christophers, Shannon N Leahy, David B Soffar, et al.
Development (Cambridge, England)|June 13, 2024
Muscle cofilin alters neuromuscular junction postsynaptic development to strengthen functional neurotransmissionBriana Christophers, Shannon N Leahy, David B Soffar, et al.
Glia|July 3, 2019
TREM2 is required for microglial instruction of astrocytic synaptic engulfment in neurodevelopmentTaylor R Jay, Victoria E von Saucken, Braulio Muñoz, et al.
Nature Communications|June 17, 2015
Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiationUlf Dettmer, Andrew J Newman, Frank Soldner, et al.
Human Molecular Genetics|September 16, 2017
Loss of native α-synuclein multimerization by strategically mutating its amphipathic helix causes abnormal vesicle interactions in neuronal cellsUlf Dettmer, Nagendran Ramalingam, Victoria E von Saucken, et al.
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