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Journal of Neuroscience Research|May 14, 2009
The Yin and Yang of Sox proteins: Activation and repression in development and diseaseLi-Jin Chew, Vittorio Gallo
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 26, 2013
Transgenic overexpression of Sox17 promotes oligodendrocyte development and attenuates demyelinationXiaotian Ming, Li-Jin Chew, Vittorio Gallo
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 20, 2010
Mechanisms of regulation of oligodendrocyte development by p38 mitogen-activated protein kinaseLi-Jin Chew, William Coley, Ying Cheng, et al.
Glia|May 28, 2005
Interferon-gamma inhibits cell cycle exit in differentiating oligodendrocyte progenitor cellsLi-Jin Chew, William C King, Ann Kennedy, et al.
Iscience|October 21, 2020
Sox17 Promotes Oligodendrocyte Regeneration by Dual Modulation of Hedgehog and Wnt SignalingXiaotian Ming, Jeffrey L Dupree, Vittorio Gallo, et al.
Cancer Letters|March 12, 2013
Modulation of the Wnt/beta-catenin pathway in human oligodendroglioma cells by Sox17 regulates proliferation and differentiationHui-Ling Chen, Li-Jin Chew, Roger J Packer, et al.
Cancer Research|March 22, 2018
Age-Dependent Cellular and Behavioral Deficits Induced by Molecularly Targeted Drugs Are ReversibleJoseph Scafidi, Jonathan Ritter, Brooke M Talbot, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 18, 2011
Cellular changes underlying hyperoxia-induced delay of white matter developmentThomas Schmitz, Jonathan Ritter, Susanne Mueller, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 24, 2013
Neonatal hyperoxia exposure disrupts axon-oligodendrocyte integrity in the subcortical white matterJonathan Ritter, Thomas Schmitz, Li-Jin Chew, et al.
Cell Reports|December 5, 2019
Sox17 Regulates a Program of Oligodendrocyte Progenitor Cell Expansion and Differentiation during Development and RepairLi-Jin Chew, Xiaotian Ming, Brian McEllin, et al.
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