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Vittorio Maglione

Showing results (1-10 of 69) with videos related to

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Frontiers in Neuroscience|May 3, 2018
Sphingolipid Metabolism: A New Therapeutic Opportunity for Brain Degenerative DisordersAlba Di Pardo, Vittorio Maglione
Trends in Pharmacological Sciences|March 22, 2018
The S1P Axis: New Exciting Route for Treating Huntington's DiseaseAlba Di Pardo, Vittorio Maglione
Frontiers in Neuroscience|October 22, 2016
Impaired Levels of Gangliosides in the Corpus Callosum of Huntington Disease Animal ModelsAlba Di Pardo, Enrico Amico, Vittorio Maglione
Journal of Neural Transmission (Vienna, Austria : 1996)|April 27, 2011
Genotype-, aging-dependent abnormal caspase activity in Huntington disease blood cellsFerdinando Squitieri, Vittorio Maglione, Sara Orobello, et al.
Frontiers in Neuroscience|October 2, 2025
Transposon activity and nucleotide triplet instability: new perspectives on their potential interplay in brain disordersGiuseppe Pepe, Marianna Storto, Alba Di Pardo, et al.
Mechanisms of Ageing and Development|November 18, 2005
Huntingtin fragmentation and increased caspase 3, 8 and 9 activities in lymphoblasts with heterozygous and homozygous Huntington's disease mutationVittorio Maglione, Milena Cannella, Roberto Gradini, et al.
International Journal of Molecular Sciences|August 6, 2020
Mitochondrial Respiration Changes in R6/2 Huntington's Disease Model Mice during Aging in a Brain Region Specific MannerJohannes Burtscher, Alba Di Pardo, Vittorio Maglione, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|November 17, 2004
New Huntington disease mutation arising from a paternal CAG34 allele showing somatic length variation in serially passaged lymphoblastsMilena Cannella, Vittorio Maglione, Tiziana Martino, et al.
Neuroscience Letters|October 14, 2005
The platelet maximum number of A2A-receptor binding sites (Bmax) linearly correlates with age at onset and CAG repeat expansion in Huntington's disease patients with predominant choreaVittorio Maglione, Milena Cannella, Tiziana Martino, et al.
Journal of Cellular and Molecular Medicine|June 23, 2015
Pridopidine, a dopamine stabilizer, improves motor performance and shows neuroprotective effects in Huntington disease R6/2 mouse modelFerdinando Squitieri, Alba Di Pardo, Mariagrazia Favellato, et al.
Pageof 7

Showing results (1-10 of 69) with videos related to

Sort By:
Pageof 7
Frontiers in Neuroscience|May 3, 2018
Sphingolipid Metabolism: A New Therapeutic Opportunity for Brain Degenerative DisordersAlba Di Pardo, Vittorio Maglione
Trends in Pharmacological Sciences|March 22, 2018
The S1P Axis: New Exciting Route for Treating Huntington's DiseaseAlba Di Pardo, Vittorio Maglione
Frontiers in Neuroscience|October 22, 2016
Impaired Levels of Gangliosides in the Corpus Callosum of Huntington Disease Animal ModelsAlba Di Pardo, Enrico Amico, Vittorio Maglione
Journal of Neural Transmission (Vienna, Austria : 1996)|April 27, 2011
Genotype-, aging-dependent abnormal caspase activity in Huntington disease blood cellsFerdinando Squitieri, Vittorio Maglione, Sara Orobello, et al.
Frontiers in Neuroscience|October 2, 2025
Transposon activity and nucleotide triplet instability: new perspectives on their potential interplay in brain disordersGiuseppe Pepe, Marianna Storto, Alba Di Pardo, et al.
Mechanisms of Ageing and Development|November 18, 2005
Huntingtin fragmentation and increased caspase 3, 8 and 9 activities in lymphoblasts with heterozygous and homozygous Huntington's disease mutationVittorio Maglione, Milena Cannella, Roberto Gradini, et al.
International Journal of Molecular Sciences|August 6, 2020
Mitochondrial Respiration Changes in R6/2 Huntington's Disease Model Mice during Aging in a Brain Region Specific MannerJohannes Burtscher, Alba Di Pardo, Vittorio Maglione, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|November 17, 2004
New Huntington disease mutation arising from a paternal CAG34 allele showing somatic length variation in serially passaged lymphoblastsMilena Cannella, Vittorio Maglione, Tiziana Martino, et al.
Neuroscience Letters|October 14, 2005
The platelet maximum number of A2A-receptor binding sites (Bmax) linearly correlates with age at onset and CAG repeat expansion in Huntington's disease patients with predominant choreaVittorio Maglione, Milena Cannella, Tiziana Martino, et al.
Journal of Cellular and Molecular Medicine|June 23, 2015
Pridopidine, a dopamine stabilizer, improves motor performance and shows neuroprotective effects in Huntington disease R6/2 mouse modelFerdinando Squitieri, Alba Di Pardo, Mariagrazia Favellato, et al.
Pageof 7