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Plos Pathogens|July 3, 2015
Herpesvirus Genome Recognition Induced Acetylation of Nuclear IFI16 Is Essential for Its Cytoplasmic Translocation, Inflammasome and IFN-β ResponsesMairaj Ahmed Ansari, Sujoy Dutta, Mohanan Valiya Veettil, et al.Journal of Virology|July 29, 2011
Deletion of Kaposi's sarcoma-associated herpesvirus FLICE inhibitory protein, vFLIP, from the viral genome compromises the activation of STAT1-responsive cellular genes and spindle cell formation in endothelial cellsKhaled R Alkharsah, Vivek Vikram Singh, Raffaella Bosco, et al.Cellular and Molecular Life Sciences : CMLS|November 18, 2016
Proliferation status defines functional properties of endothelial cellsChristoph Lipps, Muhammad Badar, Milada Butueva, et al.Plos Pathogens|October 3, 2012
Kaposi's sarcoma herpesvirus K15 protein contributes to virus-induced angiogenesis by recruiting PLCγ1 and activating NFAT1-dependent RCAN1 expressionKiran Bala, Raffaella Bosco, Silvia Gramolelli, et al.Pageof 2