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Developmental Biology|October 20, 2009
Forkhead box M1 transcriptional factor is required for smooth muscle cells during embryonic development of blood vessels and esophagusVladimir Ustiyan, I-Ching Wang, Xiaomeng Ren, et al.
Science Signaling|May 12, 2016
The transcription factor FOXF1 promotes prostate cancer by stimulating the mitogen-activated protein kinase ERK5Logan Fulford, David Milewski, Vladimir Ustiyan, et al.
Developmental Biology|August 29, 2018
FOXF1 transcription factor promotes lung morphogenesis by inducing cellular proliferation in fetal lung mesenchymeVladimir Ustiyan, Craig Bolte, Yufang Zhang, et al.
The Journal of Biological Chemistry|June 19, 2013
Foxm1 expression in prostate epithelial cells is essential for prostate carcinogenesisYuqi Cai, David Balli, Vladimir Ustiyan, et al.
Biologics : Targets & Therapy|March 27, 2023
Demonstration of Safety in Wild Type Mice of npFOXF1, a Novel Nanoparticle-Based Gene Therapy for Alveolar Capillary Dysplasia with Misaligned Pulmonary VeinsFatemeh Kohram, Zicheng Deng, Yufang Zhang, et al.
American Journal of Respiratory and Critical Care Medicine|September 3, 2020
<i>In Vivo</i> Generation of Lung and Thyroid Tissues from Embryonic Stem Cells Using Blastocyst ComplementationBingqiang Wen, Enhong Li, Vladimir Ustiyan, et al.
Disease Models & Mechanisms|December 17, 2020
Disruption of a hedgehog-foxf1-rspo2 signaling axis leads to tracheomalacia and a loss of sox9+ tracheal chondrocytesTalia Nasr, Andrea M Holderbaum, Praneet Chaturvedi, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|September 8, 2025
TGF-β induces excessive pulmonary IL-6 secretion in cystic fibrosis via PI3KGrace Scharf, Cynthia R Davidson, Vladimir Ustiyan, et al.
American Journal of Respiratory and Critical Care Medicine|June 15, 2019
The S52F FOXF1 Mutation Inhibits STAT3 Signaling and Causes Alveolar Capillary DysplasiaArun Pradhan, Andrew Dunn, Vladimir Ustiyan, et al.
American Journal of Respiratory and Critical Care Medicine|April 3, 2020
Nanoparticle Delivery of Proangiogenic Transcription Factors into the Neonatal Circulation Inhibits Alveolar Simplification Caused by HyperoxiaCraig Bolte, Vladimir Ustiyan, Xiaomeng Ren, et al.
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