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Molecular Immunology|September 24, 2025
Therapeutic targeting of neuroinflammation in sphingolipidosisEbru Ada, Volkan SeyrantepeCell and Tissue Research|February 10, 2026
Alterations in secondary lipids are associated with neuroinflammation in the brain of Neu1-deficient miceEbru Ada, Volkan SeyrantepeNeurochemistry International|March 12, 2026
Human Neuraminidase 3 reduces the severity of Tay-Sachs disease symptoms in a mouse modelHande Basırlı, Volkan SeyrantepeJournal of Molecular Medicine (Berlin, Germany)|November 8, 2024
Abnormally accumulated GM2 ganglioside contributes to skeletal deformity in Tay-Sachs miceSecil Akyildiz Demir, Volkan SeyrantepeMetabolic Brain Disease|February 28, 2025
Combined treatment of Ketogenic diet and propagermanium reduces neuroinflammation in Tay-Sachs disease mouse modelOrhan Kerim Inci, Volkan SeyrantepeArchives of Biochemistry and Biophysics|February 28, 2026
Rodent models with neuraminidase deficienciesMelike Can, Hande Basırlı, Volkan SeyrantepeMolecular Biology Reports|March 5, 2025
Imbalance in redox homeostasis is associated with neurodegeneration in the murine model of Tay-Sachs diseaseHande Basırlı, Nurselin Ateş, Volkan SeyrantepeMetabolic Brain Disease|February 25, 2026
Intrathecal delivery of AAVrh10-mHexa combined with anti-inflammatory treatment reduces neuropathological markers and extends the lifespan of mice with early-onset Tay-Sachs diseaseMelike Can, Jerome Ausseil, Volkan SeyrantepeHuman Genetics|June 20, 2006
The microcell-mediated transfer of human chromosome 8 restores the deficient N-acetylytransferase activity in skin fibroblasts of Mucopolysaccharidosis type IIIC patientsVolkan Seyrantepe, Frédérique Tihy, Alexey V PshezhetskyPlos One|March 17, 2023
Autophagic flux is impaired in the brain tissue of Tay-Sachs disease mouse modelTugce Sengul, Melike Can, Nurselin Ateş, et al.Pageof 4