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W Bodmer

Showing results (81-90 of 89) with videos related to

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Tissue Antigens|November 1, 1976
Serological identification of Ia antigens: report of a British region Ia workshopJ Bodmer, P Pickbourne, W Bodmer, et al.
Nature Medicine|September 2, 1999
The type of somatic mutation at APC in familial adenomatous polyposis is determined by the site of the germline mutation: a new facet to Knudson's 'two-hit' hypothesisH Lamlum, M Ilyas, A Rowan, et al.
Gut|August 14, 1999
Screening SMAD1, SMAD2, SMAD3, and SMAD5 for germline mutations in juvenile polyposis syndromeS Bevan, K Woodford-Richens, P Rozen, et al.
Human Molecular Genetics|September 26, 2000
Germline APC variants in patients with multiple colorectal adenomas, with evidence for the particular importance of E1317QH Lamlum, N Al Tassan, E Jaeger, et al.
Human Molecular Genetics|November 13, 1998
Mutations in DPC4 (SMAD4) cause juvenile polyposis syndrome, but only account for a minority of casesR Houlston, S Bevan, A Williams, et al.
Nature|January 15, 1998
A serine/threonine kinase gene defective in Peutz-Jeghers syndromeA Hemminki, D Markie, I Tomlinson, et al.
Journal of Medical Genetics|February 25, 1998
Peutz-Jeghers disease: most, but not all, families are compatible with linkage to 19p13.3S Olschwang, D Markie, S Seal, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 28, 2002
Whole-gene APC deletions cause classical familial adenomatous polyposis, but not attenuated polyposis or "multiple" colorectal adenomasO M Sieber, H Lamlum, M D Crabtree, et al.
Arzneimittel-Forschung|October 9, 1999
Effect of Sch 55700, a humanized monoclonal antibody to human interleukin-5, on eosinophilic responses and bronchial hyperreactivityR W Egan, D Athwal, M W Bodmer, et al.
Pageof 9

Showing results (81-90 of 89) with videos related to

Sort By:
Pageof 9
You have reached the last page of results.This site can display upto 89 results.
Tissue Antigens|November 1, 1976
Serological identification of Ia antigens: report of a British region Ia workshopJ Bodmer, P Pickbourne, W Bodmer, et al.
Nature Medicine|September 2, 1999
The type of somatic mutation at APC in familial adenomatous polyposis is determined by the site of the germline mutation: a new facet to Knudson's 'two-hit' hypothesisH Lamlum, M Ilyas, A Rowan, et al.
Gut|August 14, 1999
Screening SMAD1, SMAD2, SMAD3, and SMAD5 for germline mutations in juvenile polyposis syndromeS Bevan, K Woodford-Richens, P Rozen, et al.
Human Molecular Genetics|September 26, 2000
Germline APC variants in patients with multiple colorectal adenomas, with evidence for the particular importance of E1317QH Lamlum, N Al Tassan, E Jaeger, et al.
Human Molecular Genetics|November 13, 1998
Mutations in DPC4 (SMAD4) cause juvenile polyposis syndrome, but only account for a minority of casesR Houlston, S Bevan, A Williams, et al.
Nature|January 15, 1998
A serine/threonine kinase gene defective in Peutz-Jeghers syndromeA Hemminki, D Markie, I Tomlinson, et al.
Journal of Medical Genetics|February 25, 1998
Peutz-Jeghers disease: most, but not all, families are compatible with linkage to 19p13.3S Olschwang, D Markie, S Seal, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 28, 2002
Whole-gene APC deletions cause classical familial adenomatous polyposis, but not attenuated polyposis or "multiple" colorectal adenomasO M Sieber, H Lamlum, M D Crabtree, et al.
Arzneimittel-Forschung|October 9, 1999
Effect of Sch 55700, a humanized monoclonal antibody to human interleukin-5, on eosinophilic responses and bronchial hyperreactivityR W Egan, D Athwal, M W Bodmer, et al.
Pageof 9