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Methods and Findings in Experimental and Clinical Pharmacology|November 1, 1987
Monoclonal antibodies to canine differentiation antigensH J Deeg, W C Ladiges, J Aprile, et al.Bone Marrow Transplantation|January 1, 1990
Failure of anti-MHC antibodies to prevent GVHD in DLA mismatched unrelated canine marrow recipientsW C Ladiges, R Storb, R F Raff, et al.Immunogenetics|August 7, 1999
FVB/N (H2(q)) mouse is resistant to arthritis induction and exhibits a genomic deletion of T-cell receptor V beta gene segmentsG E Osman, M C Hannibal, J P Anderson, et al.Genetics|January 11, 2000
Cellular Werner phenotypes in mice expressing a putative dominant-negative human WRN geneL Wang, C E Ogburn, C B Ware, et al.Laboratory Animal Science|April 1, 1997
SWR: an inbred strain suitable for generating transgenic miceG E Osman, D P Jacobson, S W Li, et al.The American Journal of Pathology|June 1, 1986
Postmortem findings in four litters of dogs with familial canine dermatomyositisA M Hargis, D J Prieur, K H Haupt, et al.International Immunology|December 10, 1998
Expression of a type II collagen-specific TCR transgene accelerates the onset of arthritis in miceG E Osman, S Cheunsuk, S E Allen, et al.The American Journal of Pathology|December 10, 1998
Transgenic mice over-expressing the C-99 fragment of betaPP with an alpha-secretase site mutation develop a myopathy similar to human inclusion body myositisL W Jin, M G Hearn, C E Ogburn, et al.Immunogenetics|July 10, 1999
T-cell receptor vbeta deletion and valpha polymorphism are responsible for the resistance of SWR mouse to arthritis inductionG E Osman, M C Hannibal, J P Anderson, et al.Free Radical Biology & Medicine|October 18, 2000
Levels of DNA damage are unaltered in mice overexpressing human catalase in nucleiS E Schriner, C E Ogburn, A C Smith, et al.Pageof 4