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W Cleveland

Showing results (411-420 of 499) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|December 14, 2006
Gene transfer demonstrates that muscle is not a primary target for non-cell-autonomous toxicity in familial amyotrophic lateral sclerosisTimothy M Miller, Soo H Kim, Koji Yamanaka, et al.
Nature Communications|January 6, 2018
C9ORF72 GGGGCC repeat-associated non-AUG translation is upregulated by stress through eIF2α phosphorylationWeiwei Cheng, Shaopeng Wang, Alexander A Mestre, et al.
Developmental Cell|January 31, 2017
Centrosome Amplification Is Sufficient to Promote Spontaneous Tumorigenesis in MammalsMichelle S Levine, Bjorn Bakker, Bram Boeckx, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 16, 1994
Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activityD R Borchelt, M K Lee, H S Slunt, et al.
Nature Cell Biology|June 23, 2025
Author Correction: A two-step mechanism for epigenetic specification of centromere identity and functionDaniele Fachinetti, H Diego Folco, Yael Nechemia-Arbely, et al.
Neurology|October 1, 1996
Mechanisms of selective motor neuron death in transgenic mouse models of motor neuron diseaseD W Cleveland, L I Bruijn, P C Wong, et al.
Nature Cell Biology|July 23, 2013
A two-step mechanism for epigenetic specification of centromere identity and functionDaniele Fachinetti, H Diego Folco, Yael Nechemia-Arbely, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 20, 2007
Common molecular signature in SOD1 for both sporadic and familial amyotrophic lateral sclerosisArie Gruzman, William L Wood, Evgenia Alpert, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 31, 2002
Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS)David S Howland, Jian Liu, Yijin She, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 5, 2014
Blood-spinal cord barrier disruption contributes to early motor-neuron degeneration in ALS-model miceEthan A Winkler, Jesse D Sengillo, Abhay P Sagare, et al.
Pageof 50

Showing results (411-420 of 499) with videos related to

Sort By:
Pageof 50
Proceedings of the National Academy of Sciences of the United States of America|December 14, 2006
Gene transfer demonstrates that muscle is not a primary target for non-cell-autonomous toxicity in familial amyotrophic lateral sclerosisTimothy M Miller, Soo H Kim, Koji Yamanaka, et al.
Nature Communications|January 6, 2018
C9ORF72 GGGGCC repeat-associated non-AUG translation is upregulated by stress through eIF2α phosphorylationWeiwei Cheng, Shaopeng Wang, Alexander A Mestre, et al.
Developmental Cell|January 31, 2017
Centrosome Amplification Is Sufficient to Promote Spontaneous Tumorigenesis in MammalsMichelle S Levine, Bjorn Bakker, Bram Boeckx, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 16, 1994
Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activityD R Borchelt, M K Lee, H S Slunt, et al.
Nature Cell Biology|June 23, 2025
Author Correction: A two-step mechanism for epigenetic specification of centromere identity and functionDaniele Fachinetti, H Diego Folco, Yael Nechemia-Arbely, et al.
Neurology|October 1, 1996
Mechanisms of selective motor neuron death in transgenic mouse models of motor neuron diseaseD W Cleveland, L I Bruijn, P C Wong, et al.
Nature Cell Biology|July 23, 2013
A two-step mechanism for epigenetic specification of centromere identity and functionDaniele Fachinetti, H Diego Folco, Yael Nechemia-Arbely, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 20, 2007
Common molecular signature in SOD1 for both sporadic and familial amyotrophic lateral sclerosisArie Gruzman, William L Wood, Evgenia Alpert, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 31, 2002
Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS)David S Howland, Jian Liu, Yijin She, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 5, 2014
Blood-spinal cord barrier disruption contributes to early motor-neuron degeneration in ALS-model miceEthan A Winkler, Jesse D Sengillo, Abhay P Sagare, et al.
Pageof 50