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Cell Death and Differentiation|June 7, 2014
RIPK1- and RIPK3-induced cell death mode is determined by target availabilityW D Cook, D M Moujalled, T J Ralph, et al.Cell Death & Disease|March 1, 2014
Necroptosis induced by RIPK3 requires MLKL but not Drp1D M Moujalled, W D Cook, J M Murphy, et al.Cell Death & Disease|January 19, 2013
TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1D M Moujalled, W D Cook, T Okamoto, et al.Cell Death and Differentiation|November 19, 2011
In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent itD M Moujalled, W D Cook, J M Lluis, et al.Proceedings of the National Academy of Sciences of the United States of America|February 1, 1993
Toward an understanding of the molecular mechanisms of physiological cell deathD L VauxClinical and Experimental Pharmacology & Physiology|November 1, 1995
Hypothesis: apoptosis caused by cytotoxins represents a defensive response that evolved to combat intracellular pathogensD L Vaux, G HäckerJournal of Cell Science|May 1, 2001
Two kinds of BIR-containing protein - inhibitors of apoptosis, or required for mitosisJ Silke, D L VauxImmunology and Cell Biology|April 2, 1999
Signalling by CD95 and TNF receptors: not only life and deathC Magnusson, D L VauxBiochemistry and Cell Biology = Biochimie Et Biologie Cellulaire|November 1, 1994
Expression of candidate cell death genes in cell lines during apoptosisG Häcker, D L VauxPageof 53