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Science (New York, N.Y.)|May 5, 1995
Immune system impairment and hepatic fibrosis in mice lacking the dioxin-binding Ah receptorP Fernandez-Salguero, T Pineau, D M Hilbert, et al.Journal of Forensic Sciences|January 1, 1995
D1S80 population data in African Americans, Caucasians, southeastern Hispanics, southwestern Hispanics, and OrientalsB Budowle, F S Baechtel, J B Smerick, et al.Cell Death & Disease|January 19, 2013
TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1D M Moujalled, W D Cook, T Okamoto, et al.Proceedings of the National Academy of Sciences of the United States of America|June 24, 1997
Purification and molecular cloning of a secreted, Frizzled-related antagonist of Wnt actionP W Finch, X He, M J Kelley, et al.Nature|October 18, 1984
Molecular cloning and expression of cDNAs for the human interleukin-2 receptorW J Leonard, J M Depper, G R Crabtree, et al.Cell Death and Differentiation|June 7, 2014
RIPK1- and RIPK3-induced cell death mode is determined by target availabilityW D Cook, D M Moujalled, T J Ralph, et al.Annali Di Igiene : Medicina Preventiva E Di Comunita|December 1, 2012
Mediterranean diet pyramids: towards the Italian modelV del Balzo, L Diolordi, A Pinto, et al.The Journal of Nutrition, Health & Aging|August 15, 2003
MNA predictive value in the follow-up of geriatric patientsL M Donini, C Savina, A Rosano, et al.Cell Death and Differentiation|November 19, 2011
In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent itD M Moujalled, W D Cook, J M Lluis, et al.The EMBO Journal|January 1, 1990
Separate elements control DJ and VDJ rearrangement in a transgenic recombination substrateP Ferrier, B Krippl, T K Blackwell, et al.Pageof 30