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Biorxiv : the Preprint Server for Biology|December 15, 2025
TDP-43 suppression of ATP8A2 cryptic splicing implicates phosphatidylserine-driven neuroinflammation in ALS/FTDJames T O'Connor, Hui Qi Loo, Caiwei Guo, et al.
Acta Neuropathologica|May 2, 2025
Widespread distribution of α-synuclein oligomers in LRRK2-related Parkinson's diseaseHiroaki Sekiya, Lukas Franke, Yuki Hashimoto, et al.
Nature Neuroscience|October 21, 2025
TDP-43 nuclear loss in FTD/ALS causes widespread alternative polyadenylation changesYi Zeng, Anastasiia Lovchykova, Tetsuya Akiyama, et al.
Archives of Neurology|November 10, 2010
Functional impact of white matter hyperintensities in cognitively normal elderly subjectsMelissa E Murray, Matthew L Senjem, Ronald C Petersen, et al.
Neurology|June 11, 2020
Clinical and pathologic features of cognitive-predominant corticobasal degenerationNobutaka Sakae, Octavio A Santos, Otto Pedraza, et al.
Neurobiology of Aging|June 30, 2009
Incidental Lewy body disease: do some cases represent a preclinical stage of dementia with Lewy bodies?Roberta Frigerio, Hiroshige Fujishiro, Tae-Beom Ahn, et al.
European Journal of Neurology|June 11, 2013
Midbrain atrophy is not a biomarker of progressive supranuclear palsy pathologyJ L Whitwell, C R Jack, J E Parisi, et al.
Parkinsonism & Related Disorders|November 15, 2020
Association of mitochondrial genomic background with risk of Multiple System AtrophyRebecca R Valentino, Michael G Heckman, Patrick W Johnson, et al.
Annals of Clinical and Translational Neurology|August 27, 2019
Microglia in frontotemporal lobar degeneration with progranulin or C9ORF72 mutationsNobutaka Sakae, Shanu F Roemer, Kevin F Bieniek, et al.
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