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Radiation Research
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April 25, 2003
A biologically based model for liver cancer risk in the Swedish thorotrast patients
W F Heidenreich, Ullakarin Nyberg, P Hall
Radiation and Environmental Biophysics
|
February 1, 1997
Exact solutions of the clonal expansion model and their application to the incidence of solid tumors of atomic bomb survivors
W F Heidenreich, P Jacob, H G Paretzke
Radiation Research
|
May 16, 2001
Radiation-induced cell inactivation can increase the cancer risk
W F Heidenreich, M Atkinson, H G Paretzke
Radiation and Environmental Biophysics
|
September 28, 2005
Interaction of smoking and radon in rats: a biologically based mechanistic model
W F Heidenreich, J P Morlier, G Monchaux
Radiation and Environmental Biophysics
|
December 24, 1997
No evidence for increased tumor rates below 200 mSv in the atomic bomb survivors data
W F Heidenreich, H G Paretzke, P Jacob
Mathematical Biosciences
|
December 1, 1996
Dose timing in tumor radiotherapy: considerations of cell number stochasticity
R K Sachs, W F Heidenreich, D J Brenner
Radiation Research
|
October 28, 2006
Lung cancer risk in mice: analysis of fractionation effects and neutron RBE with a biologically motivated model
W F Heidenreich, B A Carnes, H G Paretzke
Radiation Research
|
April 9, 2013
A mechanistic model for medulloblastoma induction in mice
W F Heidenreich, A Saran, M Atkinson, et al.
Risk Analysis : an Official Publication of the Society for Risk Analysis
|
June 1, 1997
Some properties of the hazard function of the two-mutation clonal expansion model
W F Heidenreich, E G Luebeck, S H Moolgavkar
Statistics in Medicine
|
October 9, 2002
Mechanistic modelling in large case-control studies of lung cancer risk from smoking
W F Heidenreich, J Wellmann, P Jacob, et al.
Page
of 4
Search research articles
Search
Showing results (11-20 of 36) with videos related to
Sort By:
Page
of 4
Radiation Research
|
April 25, 2003
A biologically based model for liver cancer risk in the Swedish thorotrast patients
W F Heidenreich, Ullakarin Nyberg, P Hall
Radiation and Environmental Biophysics
|
February 1, 1997
Exact solutions of the clonal expansion model and their application to the incidence of solid tumors of atomic bomb survivors
W F Heidenreich, P Jacob, H G Paretzke
Radiation Research
|
May 16, 2001
Radiation-induced cell inactivation can increase the cancer risk
W F Heidenreich, M Atkinson, H G Paretzke
Radiation and Environmental Biophysics
|
September 28, 2005
Interaction of smoking and radon in rats: a biologically based mechanistic model
W F Heidenreich, J P Morlier, G Monchaux
Radiation and Environmental Biophysics
|
December 24, 1997
No evidence for increased tumor rates below 200 mSv in the atomic bomb survivors data
W F Heidenreich, H G Paretzke, P Jacob
Mathematical Biosciences
|
December 1, 1996
Dose timing in tumor radiotherapy: considerations of cell number stochasticity
R K Sachs, W F Heidenreich, D J Brenner
Radiation Research
|
October 28, 2006
Lung cancer risk in mice: analysis of fractionation effects and neutron RBE with a biologically motivated model
W F Heidenreich, B A Carnes, H G Paretzke
Radiation Research
|
April 9, 2013
A mechanistic model for medulloblastoma induction in mice
W F Heidenreich, A Saran, M Atkinson, et al.
Risk Analysis : an Official Publication of the Society for Risk Analysis
|
June 1, 1997
Some properties of the hazard function of the two-mutation clonal expansion model
W F Heidenreich, E G Luebeck, S H Moolgavkar
Statistics in Medicine
|
October 9, 2002
Mechanistic modelling in large case-control studies of lung cancer risk from smoking
W F Heidenreich, J Wellmann, P Jacob, et al.
Page
of 4