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Molecular Plant Pathology|February 23, 2018
Arabidopsis HIPP27 is a host susceptibility gene for the beet cyst nematode Heterodera schachtiiZoran S Radakovic, Muhammad Shahzad Anjam, Elizabeth Escobar, et al.
Journal of Experimental Botany|June 3, 2009
Diversity and activity of sugar transporters in nematode-induced root syncytiaJulia Hofmann, Paul H Hess, Dagmar Szakasits, et al.
Nutrients|February 13, 2019
Beta-Cryptoxanthin Inhibits Lipopolysaccharide-Induced Osteoclast Differentiation and Bone Resorption via the Suppression of Inhibitor of NF-κB Kinase ActivityNarumi Hirata, Ryota Ichimaru, Tsukasa Tominari, et al.
The Plant Journal : for Cell and Molecular Biology|July 6, 2020
Signatures of adaptation to a monocot host in the plant-parasitic cyst nematode Heterodera sacchariSomnath S Pokhare, Peter Thorpe, Pete Hedley, et al.
Nutrients|January 8, 2025
Lutein Maintains Bone Mass In Vitro and In Vivo Against Disuse-Induced Bone Loss in Hindlimb-Unloaded MiceYuki Tanaka, Tsukasa Tominari, Masaru Takatoya, et al.
Pesticide Biochemistry and Physiology|September 14, 2024
The biological activity of bacterial rhamnolipids on Arabidopsis thaliana and the cyst nematode Heterodera schachtii is linked to their molecular structureSandra Bredenbruch, Conrad Müller, Henry A Nvenankeng, et al.
The Plant Journal : for Cell and Molecular Biology|September 1, 2006
Expansins are involved in the formation of nematode-induced syncytia in roots of Arabidopsis thalianaKrzysztof Wieczorek, Bettina Golecki, Lars Gerdes, et al.
Frontiers in Plant Science|November 12, 2020
Novel Prodiginine Derivatives Demonstrate Bioactivities on Plants, Nematodes, and FungiSamer S Habash, Hannah U C Brass, Andreas S Klein, et al.
The New Phytologist|August 21, 2009
Myo-inositol oxygenase genes are involved in the development of syncytia induced by Heterodera schachtii in Arabidopsis rootsShahid Siddique, Stefanie Endres, Jamie M Atkins, et al.
The New Phytologist|October 26, 2022
Root architecture plasticity in response to endoparasitic cyst nematodes is mediated by damage signalingNina Guarneri, Jaap-Jan Willig, Mark G Sterken, et al.
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