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Mutation Research
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September 6, 2001
Susceptibility differences in chemical carcinogenesis linearize the dose-response relationship: threshold doses can be defined only for individuals
W K Lutz
Mutation Research
|
May 1, 1990
Endogenous genotoxic agents and processes as a basis of spontaneous carcinogenesis
W K Lutz
Das Offentliche Gesundheitswesen
|
May 1, 1990
[Dose-response relations in chemical carcinogenesis]
W K Lutz
Advances in Experimental Medicine and Biology
|
January 1, 1991
Dose-response relationships in chemical carcinogenesis: from DNA adducts to tumor incidence
W K Lutz
Journal of Cancer Research and Clinical Oncology
|
January 1, 1986
Quantitative evaluation of DNA binding data for risk estimation and for classification of direct and indirect carcinogens
W K Lutz
Human & Experimental Toxicology
|
February 24, 2001
A true threshold dose in chemical carcinogenesis cannot be defined for a population, irrespective of the mode of action
W K Lutz
Toxicologic Pathology
|
January 1, 1986
Endogenous formaldehyde does not produce detectable DNA-protein crosslinks in rat liver
W K Lutz
Mutation Research
|
September 28, 1998
Dose-response relationships in chemical carcinogenesis: superposition of different mechanisms of action, resulting in linear-nonlinear curves, practical thresholds, J-shapes
W K Lutz
Carcinogenesis
|
August 1, 1990
Dose-response relationship and low dose extrapolation in chemical carcinogenesis
W K Lutz
Human & Experimental Toxicology
|
January 11, 2000
Dose-response relationships in chemical carcinogenesis reflect differences in individual susceptibility. Consequences for cancer risk assessment, extrapolation, and prevention
W K Lutz
Page
of 12
Search research articles
Search
Showing results (1-10 of 120) with videos related to
Sort By:
Page
of 12
Mutation Research
|
September 6, 2001
Susceptibility differences in chemical carcinogenesis linearize the dose-response relationship: threshold doses can be defined only for individuals
W K Lutz
Mutation Research
|
May 1, 1990
Endogenous genotoxic agents and processes as a basis of spontaneous carcinogenesis
W K Lutz
Das Offentliche Gesundheitswesen
|
May 1, 1990
[Dose-response relations in chemical carcinogenesis]
W K Lutz
Advances in Experimental Medicine and Biology
|
January 1, 1991
Dose-response relationships in chemical carcinogenesis: from DNA adducts to tumor incidence
W K Lutz
Journal of Cancer Research and Clinical Oncology
|
January 1, 1986
Quantitative evaluation of DNA binding data for risk estimation and for classification of direct and indirect carcinogens
W K Lutz
Human & Experimental Toxicology
|
February 24, 2001
A true threshold dose in chemical carcinogenesis cannot be defined for a population, irrespective of the mode of action
W K Lutz
Toxicologic Pathology
|
January 1, 1986
Endogenous formaldehyde does not produce detectable DNA-protein crosslinks in rat liver
W K Lutz
Mutation Research
|
September 28, 1998
Dose-response relationships in chemical carcinogenesis: superposition of different mechanisms of action, resulting in linear-nonlinear curves, practical thresholds, J-shapes
W K Lutz
Carcinogenesis
|
August 1, 1990
Dose-response relationship and low dose extrapolation in chemical carcinogenesis
W K Lutz
Human & Experimental Toxicology
|
January 11, 2000
Dose-response relationships in chemical carcinogenesis reflect differences in individual susceptibility. Consequences for cancer risk assessment, extrapolation, and prevention
W K Lutz
Page
of 12