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W King Engel

Showing results (1-10 of 39) with videos related to

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Biochimica Et Biophysica Acta|December 3, 2014
Diagnostic histochemistry and clinical-pathological testings as molecular pathways to pathogenesis and treatment of the ageing neuromuscular system: a personal viewW King Engel
Microscopy Research and Technique|August 17, 2005
Molecular pathology and pathogenesis of inclusion-body myositisValerie Askanas, W King Engel
Neurology|January 25, 2006
Inclusion-body myositis: clinical, diagnostic, and pathologic aspectsW King Engel, Valerie Askanas
Acta Neuropathologica|November 1, 2008
Inclusion-body myositis: muscle-fiber molecular pathology and possible pathogenic significance of its similarity to Alzheimer's and Parkinson's disease brainsValerie Askanas, W King Engel
Acta Myologica : Myopathies and Cardiomyopathies : Official Journal of the Mediterranean Society of Myology|December 1, 2005
Sporadic inclusion-body myositis: a proposed key pathogenetic role of the abnormalities of the ubiquitin-proteasome system, and protein misfolding and aggregationV Askanas, W King Engel
Current Opinion in Rheumatology|October 22, 2003
Proposed pathogenetic cascade of inclusion-body myositis: importance of amyloid-beta, misfolded proteins, predisposing genes, and agingValerie Askanas, W King Engel
Neurology|January 25, 2006
Inclusion-body myositis: a myodegenerative conformational disorder associated with Abeta, protein misfolding, and proteasome inhibitionValerie Askanas, W King Engel
Current Opinion in Rheumatology|October 6, 2007
Inclusion-body myositis, a multifactorial muscle disease associated with aging: current concepts of pathogenesisValerie Askanas, W King Engel
Presse Medicale (Paris, France : 1983)|March 12, 2011
Sporadic inclusion-body myositis: conformational multifactorial ageing-related degenerative muscle disease associated with proteasomal and lysosomal inhibition, endoplasmic reticulum stress, and accumulation of amyloid-β42 oligomers and phosphorylated tauValerie Askanas, W King Engel
Current Rheumatology Reports|September 10, 2002
Newest pathogenetic considerations in inclusion-body myositis: possible role of amyloid-beta, cholesterol, relation to aging and to Alzheimer's diseaseValerie Askanas, W King Engel
Pageof 4

Showing results (1-10 of 39) with videos related to

Sort By:
Pageof 4
Biochimica Et Biophysica Acta|December 3, 2014
Diagnostic histochemistry and clinical-pathological testings as molecular pathways to pathogenesis and treatment of the ageing neuromuscular system: a personal viewW King Engel
Microscopy Research and Technique|August 17, 2005
Molecular pathology and pathogenesis of inclusion-body myositisValerie Askanas, W King Engel
Neurology|January 25, 2006
Inclusion-body myositis: clinical, diagnostic, and pathologic aspectsW King Engel, Valerie Askanas
Acta Neuropathologica|November 1, 2008
Inclusion-body myositis: muscle-fiber molecular pathology and possible pathogenic significance of its similarity to Alzheimer's and Parkinson's disease brainsValerie Askanas, W King Engel
Acta Myologica : Myopathies and Cardiomyopathies : Official Journal of the Mediterranean Society of Myology|December 1, 2005
Sporadic inclusion-body myositis: a proposed key pathogenetic role of the abnormalities of the ubiquitin-proteasome system, and protein misfolding and aggregationV Askanas, W King Engel
Current Opinion in Rheumatology|October 22, 2003
Proposed pathogenetic cascade of inclusion-body myositis: importance of amyloid-beta, misfolded proteins, predisposing genes, and agingValerie Askanas, W King Engel
Neurology|January 25, 2006
Inclusion-body myositis: a myodegenerative conformational disorder associated with Abeta, protein misfolding, and proteasome inhibitionValerie Askanas, W King Engel
Current Opinion in Rheumatology|October 6, 2007
Inclusion-body myositis, a multifactorial muscle disease associated with aging: current concepts of pathogenesisValerie Askanas, W King Engel
Presse Medicale (Paris, France : 1983)|March 12, 2011
Sporadic inclusion-body myositis: conformational multifactorial ageing-related degenerative muscle disease associated with proteasomal and lysosomal inhibition, endoplasmic reticulum stress, and accumulation of amyloid-β42 oligomers and phosphorylated tauValerie Askanas, W King Engel
Current Rheumatology Reports|September 10, 2002
Newest pathogenetic considerations in inclusion-body myositis: possible role of amyloid-beta, cholesterol, relation to aging and to Alzheimer's diseaseValerie Askanas, W King Engel
Pageof 4