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W M Armstead

Showing results (31-40 of 122) with videos related to

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Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|January 1, 1997
Role of nitric oxide, cyclic nucleotides, and the activation of ATP-sensitive K+ channels in the contribution of adenosine to hypoxia-induced pial artery dilationW M Armstead
Brain Research|December 5, 1998
Nitric oxide contributes to opioid release from glia during hypoxiaW M Armstead
The American Journal of Physiology|April 1, 1997
Role of activation of calcium-sensitive K+ channels in NO- and hypoxia-induced pial artery vasodilationW M Armstead
Journal of Neurotrauma|September 30, 1998
Brain injury impairs prostaglandin cerebrovasodilationW M Armstead
Brain Research|February 7, 1997
Role of activation of calcium-sensitive K+ channels and cAMP in opioid-induced pial artery dilationW M Armstead
Brain Research|June 8, 2000
NOC/oFQ contributes to hypoxic-ischemic impairment of N-methyl-D-aspartate-induced cerebral vasodilationW M Armstead
Brain Research|October 20, 1999
Endothelin-1 contributes to normocapnic hyperoxic pial artery vasoconstrictionW M Armstead
The American Journal of Physiology|November 24, 1999
Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injuryW M Armstead
Brain Research Bulletin|February 17, 2001
Role of altered cyclooxygenase metabolism in impaired cerebrovasodilation to nociceptin/orphanin FQ following brain injuryW M Armstead
Journal of Neurotrauma|July 5, 2001
Age-Dependent vasopressinergic modulation of Noc/oFQ-induced impairment of NMDA cerebrovasodilation after brain injuryW M Armstead
Pageof 13

Showing results (31-40 of 122) with videos related to

Sort By:
Pageof 13
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|January 1, 1997
Role of nitric oxide, cyclic nucleotides, and the activation of ATP-sensitive K+ channels in the contribution of adenosine to hypoxia-induced pial artery dilationW M Armstead
Brain Research|December 5, 1998
Nitric oxide contributes to opioid release from glia during hypoxiaW M Armstead
The American Journal of Physiology|April 1, 1997
Role of activation of calcium-sensitive K+ channels in NO- and hypoxia-induced pial artery vasodilationW M Armstead
Journal of Neurotrauma|September 30, 1998
Brain injury impairs prostaglandin cerebrovasodilationW M Armstead
Brain Research|February 7, 1997
Role of activation of calcium-sensitive K+ channels and cAMP in opioid-induced pial artery dilationW M Armstead
Brain Research|June 8, 2000
NOC/oFQ contributes to hypoxic-ischemic impairment of N-methyl-D-aspartate-induced cerebral vasodilationW M Armstead
Brain Research|October 20, 1999
Endothelin-1 contributes to normocapnic hyperoxic pial artery vasoconstrictionW M Armstead
The American Journal of Physiology|November 24, 1999
Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injuryW M Armstead
Brain Research Bulletin|February 17, 2001
Role of altered cyclooxygenase metabolism in impaired cerebrovasodilation to nociceptin/orphanin FQ following brain injuryW M Armstead
Journal of Neurotrauma|July 5, 2001
Age-Dependent vasopressinergic modulation of Noc/oFQ-induced impairment of NMDA cerebrovasodilation after brain injuryW M Armstead
Pageof 13