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W M Armstead

Showing results (41-50 of 122) with videos related to

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American Journal of Physiology. Heart and Circulatory Physiology|November 22, 2000
NOC/oFQ PKC-dependent superoxide generation contributes to hypoxic-ischemic impairment of NMDA cerebrovasodilationW M Armstead
Stroke|June 2, 2001
Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injuryW M Armstead
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|May 1, 1995
The contribution of delta 1- and delta 2-opioid receptors to hypoxia-induced pial artery dilation in the newborn pigW M Armstead
The American Journal of Physiology|April 1, 1996
Influence of brain injury on vasopressin-induced pial artery vasodilation: role of superoxide anionW M Armstead
The American Journal of Physiology|September 2, 1998
Relationship among NO, the KATP channel, and opioids in hypoxic pial artery dilationW M Armstead
The American Journal of Physiology|September 1, 1995
Opioids contribute to hypoxia-induced pial artery dilation through activation of ATP-sensitive K+ channelsV Shankar, W M Armstead
Stroke|August 6, 2000
Superoxide generation links nociceptin/orphanin FQ (NOC/oFQ) release to impaired N-methyl-D-aspartate cerebrovasodilation after brain injuryM Kulkarni, W M Armstead
Brain Research|January 3, 2001
Vasopressin impairs K(ATP) and K(ca) channel function after brain injuryA Salvucci, W M Armstead
The American Journal of Physiology|December 9, 1998
Vasopressin contributes to dynorphin modulation of hypoxic cerebrovasodilationA Venteicher, W M Armstead
Brain Research Bulletin|September 7, 2001
Nociceptin/orphanin FQ contributes to hypoxic/ischemic impairment of hypercapnic cerebrovasodilationA Jagolino, W M Armstead
Pageof 13

Showing results (41-50 of 122) with videos related to

Sort By:
Pageof 13
American Journal of Physiology. Heart and Circulatory Physiology|November 22, 2000
NOC/oFQ PKC-dependent superoxide generation contributes to hypoxic-ischemic impairment of NMDA cerebrovasodilationW M Armstead
Stroke|June 2, 2001
Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injuryW M Armstead
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|May 1, 1995
The contribution of delta 1- and delta 2-opioid receptors to hypoxia-induced pial artery dilation in the newborn pigW M Armstead
The American Journal of Physiology|April 1, 1996
Influence of brain injury on vasopressin-induced pial artery vasodilation: role of superoxide anionW M Armstead
The American Journal of Physiology|September 2, 1998
Relationship among NO, the KATP channel, and opioids in hypoxic pial artery dilationW M Armstead
The American Journal of Physiology|September 1, 1995
Opioids contribute to hypoxia-induced pial artery dilation through activation of ATP-sensitive K+ channelsV Shankar, W M Armstead
Stroke|August 6, 2000
Superoxide generation links nociceptin/orphanin FQ (NOC/oFQ) release to impaired N-methyl-D-aspartate cerebrovasodilation after brain injuryM Kulkarni, W M Armstead
Brain Research|January 3, 2001
Vasopressin impairs K(ATP) and K(ca) channel function after brain injuryA Salvucci, W M Armstead
The American Journal of Physiology|December 9, 1998
Vasopressin contributes to dynorphin modulation of hypoxic cerebrovasodilationA Venteicher, W M Armstead
Brain Research Bulletin|September 7, 2001
Nociceptin/orphanin FQ contributes to hypoxic/ischemic impairment of hypercapnic cerebrovasodilationA Jagolino, W M Armstead
Pageof 13