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Proceedings of the National Academy of Sciences of the United States of America|June 6, 1995
Scavenger receptor A gene regulatory elements target gene expression to macrophages and to foam cells of atherosclerotic lesionsA Horvai, W Palinski, H Wu, et al.Lancet (London, England)|October 16, 1999
Influence of maternal hypercholesterolaemia during pregnancy on progression of early atherosclerotic lesions in childhood: Fate of Early Lesions in Children (FELIC) studyC Napoli, C K Glass, J L Witztum, et al.Circulation|April 20, 1999
Intracranial arteries of human fetuses are more resistant to hypercholesterolemia-induced fatty streak formation than extracranial arteriesC Napoli, J L Witztum, F de Nigris, et al.Journal of Lipid Research|June 4, 1998
Effect of probucol on LDL oxidation and atherosclerosis in LDL receptor-deficient miceD A Bird, R K Tangirala, J Fruebis, et al.Journal of Nuclear Cardiology : Official Publication of the American Society of Nuclear Cardiology|March 10, 1999
Radiolabeled MDA2, an oxidation-specific, monoclonal antibody, identifies native atherosclerotic lesions in vivoS Tsimikas, W Palinski, S E Halpern, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|May 1, 1995
Immunological evidence for the presence of advanced glycosylation end products in atherosclerotic lesions of euglycemic rabbitsW Palinski, T Koschinsky, S W Butler, et al.The Journal of Clinical Investigation|January 1, 1991
Macrophage-derived foam cells freshly isolated from rabbit atherosclerotic lesions degrade modified lipoproteins, promote oxidation of low-density lipoproteins, and contain oxidation-specific lipid-protein adductsM E Rosenfeld, J C Khoo, E Miller, et al.Blood|January 12, 2001
Circulating autoantibodies to oxidized cardiolipin correlate with isoprostane F(2alpha)-VI levels and the extent of atherosclerosis in ApoE-deficient mice: modulation by vitamin ED Praticò, R K Tangirala, S Hörkkö, et al.The Journal of Clinical Investigation|February 12, 1998
Fatty streak formation occurs in human fetal aortas and is greatly enhanced by maternal hypercholesterolemia. Intimal accumulation of low density lipoprotein and its oxidation precede monocyte recruitment into early atherosclerotic lesionsC Napoli, F P D'Armiento, F P Mancini, et al.Arteriosclerosis and Thrombosis : a Journal of Vascular Biology|April 1, 1994
ApoE-deficient mice are a model of lipoprotein oxidation in atherogenesis. Demonstration of oxidation-specific epitopes in lesions and high titers of autoantibodies to malondialdehyde-lysine in serumW Palinski, V A Ord, A S Plump, et al.Pageof 8