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The Journal of Biological Chemistry|April 6, 2024
Selenoprotein I is indispensable for ether lipid homeostasis and proper myelinationLance G A Nunes, Chi Ma, FuKun W Hoffmann, et al.
The Journal of Biological Chemistry|July 25, 2013
Deletion of selenoprotein M leads to obesity without cognitive deficitsMatthew W Pitts, Mariclair A Reeves, Ann C Hashimoto, et al.
Frontiers in Nutrition|May 24, 2021
Juvenile Selenium Deficiency Impairs Cognition, Sensorimotor Gating, and Energy Homeostasis in MiceVictor W Kilonzo, Alexandru R Sasuclark, Daniel J Torres, et al.
Genes, Brain, and Behavior|April 11, 2012
Absence of selenoprotein P but not selenocysteine lyase results in severe neurological dysfunctionA V Raman, M W Pitts, A Seyedali, et al.
Journal of Leukocyte Biology|August 2, 2022
Selenoprotein I deficiency in T cells promotes differentiation into tolerant phenotypes while decreasing Th17 pathologyChi Ma, FuKun W Hoffmann, Lance G Nunes, et al.
Magnetic Resonance in Medicine|December 1, 1987
19F magnetic resonance imaging of the reticuloendothelial systemA V Ratner, R Hurd, H H Muller, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 21, 2015
Competition between the Brain and Testes under Selenium-Compromised Conditions: Insight into Sex Differences in Selenium Metabolism and Risk of Neurodevelopmental DiseaseMatthew W Pitts, Penny M Kremer, Ann C Hashimoto, et al.
AIDS Research and Human Retroviruses|September 1, 1991
Soluble CD4-PE40 is cytotoxic for a transfected mammalian cell line stably expressing the envelope protein of human immunodeficiency virus (HIV-1), and cytotoxicity is variably inhibited by the sera of HIV-1-infected patientsT W Pitts, M J Bohanon, M F Leach, et al.
The Journal of Biological Chemistry|March 1, 2021
Disrupted glucose homeostasis and skeletal-muscle-specific glucose uptake in an exocyst knockout mouse modelBrent A Fujimoto, Madison Young, Nicole Nakamura, et al.
Antioxidants (Basel, Switzerland)|February 25, 2023
Selenium Protects Mouse Hypothalamic Cells from Glucocorticoid-Induced Endoplasmic Reticulum Stress Vulnerability and Insulin Signaling ImpairmentKatlyn J An, Ashley N Hanato, Katherine W Hui, et al.
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