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The Journal of Biological Chemistry|July 23, 2003
A novel role for RAX, the cellular activator of PKR, in synergistically stimulating SV40 large T antigen-dependent gene expressionMingli Yang, Takahiko Ito, W Stratford MayBlood|October 11, 2008
Protein phosphatase 2A inactivates Bcl2's antiapoptotic function by dephosphorylation and up-regulation of Bcl2-p53 bindingXingming Deng, Fengqin Gao, W Stratford MayBlood|June 21, 2003
Cleavage of Bax to p18 Bax accelerates stress-induced apoptosis, and a cathepsin-like protease may rapidly degrade p18 BaxXuefang Cao, Xingming Deng, W Stratford MayBlood|July 12, 2003
Bcl2 retards G1/S cell cycle transition by regulating intracellular ROSXingming Deng, Fengqin Gao, W Stratford MayThe Journal of Biological Chemistry|April 1, 2008
c-Myc and caspase-2 are involved in activating Bax during cytotoxic drug-induced apoptosisXuefang Cao, Richard L Bennett, W Stratford MayCell Cycle (Georgetown, Tex.)|July 1, 2011
JAZ mediates G1 cell cycle arrest by interacting with and inhibiting E2F1Mingli Yang, Song Wu, Jinghua Jia, et al.The Journal of Biological Chemistry|August 10, 2004
Serine 18 phosphorylation of RAX, the PKR activator, is required for PKR activation and consequent translation inhibitionRichard L Bennett, William L Blalock, W Stratford MayProceedings of the National Academy of Sciences of the United States of America|December 9, 2003
Mono- and multisite phosphorylation enhances Bcl2's antiapoptotic function and inhibition of cell cycle entry functionsXingming Deng, Fengqin Gao, Tammy Flagg, et al.Blood|August 26, 2006
JAZ mediates G1 cell-cycle arrest and apoptosis by positively regulating p53 transcriptional activityMingli Yang, Song Wu, Xuekun Su, et al.Neoplasia (New York, N.Y.)|September 16, 2014
Progressive genomic instability in the Nup98-HoxD13 model of MDS correlates with loss of the PIG-A gene productMichael Byrne, Richard L Bennett, Xiaodong Cheng, et al.Pageof 4