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Cell Death and Differentiation|December 10, 1998
Rel blocks both anti-Fas- and TNF alpha-induced apoptosis and an intact Rel transactivation domain is essential for this effectW X Zong, J Bash, C GélinasMolecular and Cellular Biology|October 29, 1997
c-Rel arrests the proliferation of HeLa cells and affects critical regulators of the G1/S-phase transitionJ Bash, W X Zong, C GélinasOncogene|August 18, 1997
v-Rel prevents apoptosis in transformed lymphoid cells and blocks TNFalpha-induced cell deathW X Zong, M Farrell, J Bash, et al.The Journal of Biological Chemistry|October 27, 1995
The Ser36-Ser37 pair in HeLa nuclear protein p21/SIIR mediates Ser/Thr phosphorylation and is essential for Rous sarcoma virus long terminal repeat repressionC H Yeh, W X Zong, A J ShatkinOncogene|March 14, 2000
N-terminal determinants of I kappa B alpha necessary for the cytoplasmic regulation of c-RelI Luque, W X Zong, C Chen, et al.Genes & Development|February 27, 1999
The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosisW X Zong, L C Edelstein, C Chen, et al.Oncogene|August 29, 2007
Bfl-1/A1 functions, similar to Mcl-1, as a selective tBid and Bak antagonistM J Simmons, G Fan, W-X Zong, et al.Genes & Development|June 19, 2001
BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and BakW X Zong, T Lindsten, A J Ross, et al.The EMBO Journal|May 18, 1999
Rel/NF-kappaB can trigger the Notch signaling pathway by inducing the expression of Jagged1, a ligand for Notch receptorsJ Bash, W X Zong, S Banga, et al.Cold Spring Harbor Symposia on Quantitative Biology|July 28, 2006
How do cancer cells acquire the fuel needed to support cell growth?C B Thompson, D E Bauer, J J Lum, et al.Pageof 2