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EMBO Molecular Medicine|September 7, 2022
Neuropilin-1 (NRP1) expression distinguishes self-reactive helper T cells in systemic autoimmune diseaseBen Je Raveney, Yosif El-Darawish, Wakiro Sato, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 16, 2011
Interleukin 6 signaling promotes anti-aquaporin 4 autoantibody production from plasmablasts in neuromyelitis opticaNorio Chihara, Toshimasa Aranami, Wakiro Sato, et al.
BMJ Neurology Open|June 17, 2024
'Grasshopper sign': the novel imaging of post-COVID-19 myelopathy with delayed longitudinal white matter abnormalitiesMotohiro Okumura, Kazumasa Sekiguchi, Tomoko Okamoto, et al.
Neurology(R) Neuroimmunology & Neuroinflammation|February 13, 2024
CD11c<sup>high</sup> B Cell Expansion Is Associated With Severity and Brain Atrophy in Neuromyelitis OpticaEiichiro Amano, Wakiro Sato, Yukio Kimura, et al.
Neurology(R) Neuroimmunology & Neuroinflammation|March 24, 2016
Disrupted balance of T cells under natalizumab treatment in multiple sclerosisKimitoshi Kimura, Masakazu Nakamura, Wakiro Sato, et al.
International Immunology|August 28, 2025
The thyroid hormone receptor beta (TR-β) signaling controls pathogenic Th17 cells in autoimmune diseaseYoshimitsu Doi, Ben J E Raveney, Atsuko Kimura, et al.
Nature Communications|January 4, 2018
Circulating exosomes suppress the induction of regulatory T cells via let-7i in multiple sclerosisKimitoshi Kimura, Hirohiko Hohjoh, Masashi Fukuoka, et al.
Annals of Neurology|August 23, 2021
Th1 - CD11c<sup>+</sup> B Cell Axis Associated with Response to Plasmapheresis in Multiple SclerosisKimitoshi Kimura, Youwei Lin, Hiromi Yamaguchi, et al.
JMIR Research Protocols|January 15, 2024
Efficacy and Safety of the Natural Killer T Cell-Stimulatory Glycolipid OCH-NCNP1 for Patients With Relapsing Multiple Sclerosis: Protocol for a Randomized Placebo-Controlled Clinical TrialTomoko Okamoto, Takami Ishizuka, Reiko Shimizu, et al.
Journal of Human Genetics|October 11, 2018
PLA2G6-associated neurodegeneration presenting as a complicated form of hereditary spastic paraplegiaKishin Koh, Yuta Ichinose, Hiroyuki Ishiura, et al.
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