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Molecular Cancer Therapeutics|March 5, 2022
Optimal Strategy and Benefit of Pulsed Therapy Depend On Tumor Heterogeneity and Aggressiveness at Time of Treatment InitiationDeepti Mathur, Bradford P Taylor, Walid K Chatila, et al.
Cancers|December 10, 2021
Early TP53 Alterations Shape Gastric and Esophageal Cancer DevelopmentPranshu Sahgal, Brandon M Huffman, Deepa T Patil, et al.
Journal of the National Comprehensive Cancer Network : JNCCN|February 5, 2021
Tumor Mutational Burden and Mismatch Repair Deficiency Discordance as a Mechanism of Immunotherapy ResistanceAgata A Bielska, Walid K Chatila, Henry Walch, et al.
Nature Communications|August 17, 2019
Cancer-associated mutations in DICER1 RNase IIIa and IIIb domains exert similar effects on miRNA biogenesisJeffrey Vedanayagam, Walid K Chatila, Bülent Arman Aksoy, et al.
Nature Metabolism|April 9, 2021
Respiratory complex and tissue lineage drive recurrent mutations in tumour mtDNAAlexander N Gorelick, Minsoo Kim, Walid K Chatila, et al.
Modern Pathology : an Official Journal of the United States and Canadian Academy of Pathology, Inc|June 26, 2020
Lung-only melanoma: UV mutational signature supports origin from occult cutaneous primaries and argues against the concept of primary pulmonary melanomaChen Yang, Francisco Sanchez-Vega, Jason C Chang, et al.
Annals of Surgical Oncology|August 27, 2020
Primary Tumor Location and Outcomes After Cytoreductive Surgery and Intraperitoneal Chemotherapy for Peritoneal Metastases of Colorectal OriginMohammad Adileh, Jonathan B Yuval, Henry S Walch, et al.
Oncogene|August 16, 2024
Cooperative genomic lesions in HRAS-mutant cancers predict resistance to farnesyltransferase inhibitorsAradhya Nigam, Gnana P Krishnamoorthy, Walid K Chatila, et al.
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