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Journal of Neuroinflammation|June 22, 2018
Vitexin reduces epilepsy after hypoxic ischemia in the neonatal brain via inhibition of NKCC1Wen-di Luo, Jia-Wei Min, Wen-Xian Huang, et al.CNS Neuroscience & Therapeutics|March 27, 2018
A Kv1.3 channel-specific blocker alleviates neurological impairment through inhibiting T-cell activation in experimental autoimmune encephalomyelitisXiao-Lu Yuan, Yi-Peng Zhao, Jie Huang, et al.Journal of Neuroinflammation|May 31, 2019
TRPV1 mediates astrocyte activation and interleukin-1β release induced by hypoxic ischemia (HI)Xing-Liang Yang, Xin Wang, Lin Shao, et al.Frontiers in Cell and Developmental Biology|January 11, 2020
NFAT5 and HIF-1α Coordinate to Regulate NKCC1 Expression in Hippocampal Neurons After Hypoxia-IschemiaXing-Liang Yang, Meng-Liu Zeng, Lin Shao, et al.Brain, Behavior, and Immunity|March 25, 2015
TRPV1 promotes repetitive febrile seizures by pro-inflammatory cytokines in immature brainWen-Xian Huang, Fang Yu, Russell M Sanchez, et al.Journal of Neuroinflammation|November 15, 2019
TRPV1 translocated to astrocytic membrane to promote migration and inflammatory infiltration thus promotes epilepsy after hypoxic ischemia in immature brainXin Wang, Xing-Liang Yang, Wei-Lin Kong, et al.Neuropharmacology|July 19, 2015
Vitexin reduces hypoxia-ischemia neonatal brain injury by the inhibition of HIF-1alpha in a rat pup modelJia-Wei Min, Jiang-Jian Hu, Miao He, et al.Neuroscience Bulletin|July 26, 2021
Complement C3 Aggravates Post-epileptic Neuronal Injury Via Activation of TRPV1Guang-Tong Jiang, Lin Shao, Shuo Kong, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|January 8, 2021
Silencing of circIgf1r plays a protective role in neuronal injury via regulating astrocyte polarization during epilepsyLin Shao, Guang-Tong Jiang, Xing-Liang Yang, et al.Journal of Neuroinflammation|October 26, 2023
Cell-specific NFIA upregulation promotes epileptogenesis by TRPV4-mediated astrocyte reactivityShuo Kong, Tao-Xiang Chen, Xiang-Lei Jia, et al.Pageof 3