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Kidney International|October 22, 2004
Combining TGF-beta inhibition and angiotensin II blockade results in enhanced antifibrotic effectLing Yu, Wayne A Border, Ian Anderson, et al.American Journal of Physiology. Renal Physiology|April 27, 2012
Receptor-mediated nonproteolytic activation of prorenin and induction of TGF-β1 and PAI-1 expression in renal mesangial cellsJiandong Zhang, Jie Wu, Chunyan Gu, et al.The Journal of Clinical Investigation|August 5, 2003
A mutant, noninhibitory plasminogen activator inhibitor type 1 decreases matrix accumulation in experimental glomerulonephritisYufeng Huang, Masashi Haraguchi, Daniel A Lawrence, et al.Kidney International|March 13, 2003
L-arginine supplementation accelerates renal fibrosis and shortens life span in experimental lupus nephritisHarm Peters, Wayne A Border, Matthias Rückert, et al.Journal of the American Society of Nephrology : JASN|January 25, 2008
A PAI-1 mutant, PAI-1R, slows progression of diabetic nephropathyYufeng Huang, Wayne A Border, Ling Yu, et al.American Journal of Physiology. Renal Physiology|March 28, 2008
Aldosterone and TGF-beta1 synergistically increase PAI-1 and decrease matrix degradation in rat renal mesangial and fibroblast cellsWei Huang, Chen Xu, Kyoung W Kahng, et al.Journal of the Renin-Angiotensin-Aldosterone System : JRAAS|December 11, 2012
Targeting reduction of proteinuria in glomerulonephritis: Maximizing the antifibrotic effect of valsartan by protecting podocytesChunyan Gu, Guangyu Zhou, Nancy A Noble, et al.Journal of the American Society of Nephrology : JASN|August 26, 2003
Genes expressed by the kidney, but not by bone marrow-derived cells, underlie the genetic predisposition to progressive glomerulosclerosis after mesangial injuryJoris A Aben, Dennis A Hoogervorst, Leendert C Paul, et al.Pageof 2