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American Journal of Physiology. Endocrinology and Metabolism|December 24, 2009
Cardiomyocyte-derived adiponectin is biologically active in protecting against myocardial ischemia-reperfusion injuryYajing Wang, Wayne Bond Lau, Erhe Gao, et al.
Experimental and Therapeutic Medicine|December 3, 2014
Adiponectin protects against paraquat-induced lung injury by attenuating oxidative/nitrative stressRong Yao, Yaxiong Zhou, Yarong He, et al.
Basic Research in Cardiology|December 6, 2012
Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic miceHui Su, Yuexing Yuan, Xiao-Ming Wang, et al.
American Journal of Physiology. Endocrinology and Metabolism|March 19, 2015
C1q-TNF-related protein-9, a novel cardioprotetcive cardiokine, requires proteolytic cleavage to generate a biologically active globular domain isoformYuexing Yuan, Wayne Bond Lau, Hui Su, et al.
American Journal of Physiology. Endocrinology and Metabolism|May 28, 2009
Cardioprotective effect of adiponectin is partially mediated by its AMPK-independent antinitrative actionYajing Wang, Ling Tao, Yuexin Yuan, et al.
European Heart Journal|February 7, 2012
Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injuryJun Pu, Ancai Yuan, Peiren Shan, et al.
Cancer Letters|December 20, 2014
MicroRNAs in colorectal cancer: small molecules with big functionsYu Xuan, Huiliang Yang, Linjie Zhao, et al.
Medicine|December 31, 2015
Low-Dose Versus Standard-Dose Tissue Plasminogen Activator in Acute Ischemic Stroke in Asian Populations: A Meta-AnalysisMeng-Dong Liu, Wei-Dong Ning, Ren-Cong Wang, et al.
Plos One|September 24, 2013
Inhibition of Fas-associated death domain-containing protein (FADD) protects against myocardial ischemia/reperfusion injury in a heart failure mouse modelQian Fan, Zheng M Huang, Matthieu Boucher, et al.
Cardiovascular Drugs and Therapy|February 13, 2019
Activation of T Lymphocytes as a Novel Mechanism in Beta1-Adrenergic Receptor Autoantibody-Induced Cardiac RemodelingYunhui Du, Xiao Li, Haicun Yu, et al.
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