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Neurobiology of Disease|September 15, 2011
Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortexSophie Sokolow, Sanh H Luu, Karabi Nandy, et al.
Acta Neuropathologica Communications|December 3, 2017
Cerebrovascular pathology in Down syndrome and Alzheimer diseaseElizabeth Head, Michael J Phelan, Eric Doran, et al.
The Journal of Biological Chemistry|March 29, 2005
Coq3 and Coq4 define a polypeptide complex in yeast mitochondria for the biosynthesis of coenzyme QBeth Marbois, Peter Gin, Kym F Faull, et al.
ACS Chemical Neuroscience|December 21, 2013
Quantitative analysis of axonal transport by using compartmentalized and surface micropatterned culture of neuronsHyung Joon Kim, Jeong Won Park, Jae Hwan Byun, et al.
Brain Pathology (Zurich, Switzerland)|April 11, 2012
Extensive p-tau pathology and SDS-stable p-tau oligomers in Alzheimer's cortical synapsesKristen M Henkins, Sophie Sokolow, Carol A Miller, et al.
Journal of Neurochemistry|November 14, 2014
Pre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's diseaseSophie Sokolow, Kristen M Henkins, Tina Bilousova, et al.
Acta Neuropathologica Communications|June 25, 2016
Increased tauopathy drives microglia-mediated clearance of beta-amyloidWesley Chen, Edsel A Abud, Stephen T Yeung, et al.
The Journal of Biological Chemistry|April 20, 2013
β-Amyloid (Aβ) oligomers impair brain-derived neurotrophic factor retrograde trafficking by down-regulating ubiquitin C-terminal hydrolase, UCH-L1Wayne W Poon, Anthony J Carlos, Brittany L Aguilar, et al.
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