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Genes & Development|January 5, 2006
Necrotic death as a cell fateWei-Xing Zong, Craig B Thompson
The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry|January 6, 2005
Defining the role of the Bcl-2 family of proteins in the nervous systemTullia Lindsten, Wei-Xing Zong, Craig B Thompson
The Journal of Biological Chemistry|April 14, 2007
Activation of poly(ADP)-ribose polymerase (PARP-1) induces release of the pro-inflammatory mediator HMGB1 from the nucleusDara Ditsworth, Wei-Xing Zong, Craig B Thompson
Nature Immunology|September 24, 2002
Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasisJeffrey C Rathmell, Tullia Lindsten, Wei-Xing Zong, et al.
Genes & Development|May 18, 2004
Alkylating DNA damage stimulates a regulated form of necrotic cell deathWei-Xing Zong, Dara Ditsworth, Daniel E Bauer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 6, 2003
The proapoptotic activities of Bax and Bak limit the size of the neural stem cell poolTullia Lindsten, Jeffrey A Golden, Wei-Xing Zong, et al.
The Journal of Cell Biology|July 9, 2003
Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosisWei-Xing Zong, Chi Li, Georgia Hatzivassiliou, et al.
Oncogene|August 7, 2003
The proteasome inhibitor PS-341 overcomes TRAIL resistance in Bax and caspase 9-negative or Bcl-xL overexpressing cellsThomas R Johnson, Kimberley Stone, Malti Nikrad, et al.
Cancer Biology & Therapy|January 17, 2004
Histone, H1.2: another housekeeping protein that killsWei-Xing Zong
Molecular and Cellular Biology|June 8, 2002
The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinaseKui Lei, Anjaruwee Nimnual, Wei-Xing Zong, et al.
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