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Weili Cai

Showing results (21-30 of 42) with videos related to

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Journal of Proteome Research|October 29, 2025
RSK Inhibition Rewires Kinase Networks and Triggers Cascading Pathway Disruption in Esophageal CancerLei Cheng, Haochen Xu, Tingyan Wu, et al.
Journal of Cell Science|January 17, 2012
The epigenetic H3S10 phosphorylation mark is required for counteracting heterochromatic spreading and gene silencing in Drosophila melanogasterChao Wang, Weili Cai, Yeran Li, et al.
The Journal of Biological Chemistry|September 30, 2008
The COOH-terminal domain of the JIL-1 histone H3S10 kinase interacts with histone H3 and is required for correct targeting to chromatinXiaomin Bao, Weili Cai, Huai Deng, et al.
Genetics|May 12, 2010
JIL-1 and Su(var)3-7 interact genetically and counteract each other's effect on position-effect variegation in DrosophilaHuai Deng, Weili Cai, Chao Wang, et al.
Plos One|May 3, 2013
Evidence against a role for the JIL-1 kinase in H3S28 phosphorylation and 14-3-3 recruitment to active genes in DrosophilaChao Wang, Changfu Yao, Yeran Li, et al.
Oncotarget|August 29, 2015
Inhibition of poly(ADP-ribose) polymerase 1 protects against acute myeloid leukemia by suppressing the myeloproliferative leukemia virus oncogeneLingbo Wang, Weili Cai, Wei Zhang, et al.
Immunity, Inflammation and Disease|June 27, 2022
CHD4 orchestrates the symphony of T and B lymphocytes development and a good mediator in preventing from autoimmune diseaseMiaomiao Jia, Xueqin Zou, Shuying Yin, et al.
Development (Cambridge, England)|January 18, 2008
Ectopic histone H3S10 phosphorylation causes chromatin structure remodeling in DrosophilaHuai Deng, Xiaomin Bao, Weili Cai, et al.
The Journal of Biological Chemistry|June 1, 2013
Domain requirements of the JIL-1 tandem kinase for histone H3 serine 10 phosphorylation and chromatin remodeling in vivoYeran Li, Weili Cai, Chao Wang, et al.
Development (Cambridge, England)|August 16, 2017
H2Av facilitates H3S10 phosphorylation but is not required for heat shock-induced chromatin decondensation or transcriptional elongationYeran Li, Chao Wang, Weili Cai, et al.
Pageof 5

Showing results (21-30 of 42) with videos related to

Sort By:
Pageof 5
Journal of Proteome Research|October 29, 2025
RSK Inhibition Rewires Kinase Networks and Triggers Cascading Pathway Disruption in Esophageal CancerLei Cheng, Haochen Xu, Tingyan Wu, et al.
Journal of Cell Science|January 17, 2012
The epigenetic H3S10 phosphorylation mark is required for counteracting heterochromatic spreading and gene silencing in Drosophila melanogasterChao Wang, Weili Cai, Yeran Li, et al.
The Journal of Biological Chemistry|September 30, 2008
The COOH-terminal domain of the JIL-1 histone H3S10 kinase interacts with histone H3 and is required for correct targeting to chromatinXiaomin Bao, Weili Cai, Huai Deng, et al.
Genetics|May 12, 2010
JIL-1 and Su(var)3-7 interact genetically and counteract each other's effect on position-effect variegation in DrosophilaHuai Deng, Weili Cai, Chao Wang, et al.
Plos One|May 3, 2013
Evidence against a role for the JIL-1 kinase in H3S28 phosphorylation and 14-3-3 recruitment to active genes in DrosophilaChao Wang, Changfu Yao, Yeran Li, et al.
Oncotarget|August 29, 2015
Inhibition of poly(ADP-ribose) polymerase 1 protects against acute myeloid leukemia by suppressing the myeloproliferative leukemia virus oncogeneLingbo Wang, Weili Cai, Wei Zhang, et al.
Immunity, Inflammation and Disease|June 27, 2022
CHD4 orchestrates the symphony of T and B lymphocytes development and a good mediator in preventing from autoimmune diseaseMiaomiao Jia, Xueqin Zou, Shuying Yin, et al.
Development (Cambridge, England)|January 18, 2008
Ectopic histone H3S10 phosphorylation causes chromatin structure remodeling in DrosophilaHuai Deng, Xiaomin Bao, Weili Cai, et al.
The Journal of Biological Chemistry|June 1, 2013
Domain requirements of the JIL-1 tandem kinase for histone H3 serine 10 phosphorylation and chromatin remodeling in vivoYeran Li, Weili Cai, Chao Wang, et al.
Development (Cambridge, England)|August 16, 2017
H2Av facilitates H3S10 phosphorylation but is not required for heat shock-induced chromatin decondensation or transcriptional elongationYeran Li, Chao Wang, Weili Cai, et al.
Pageof 5