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Wen-Jin Wu

Showing results (1-10 of 81) with videos related to

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Nature Reviews. Cardiology|September 30, 2015
Trastuzumab is not a tyrosine kinase inhibitorNishant Mohan, Wen Jin Wu
Advances in Clinical Chemistry|August 2, 2015
Monitoring Trastuzumab Resistance and Cardiotoxicity: A Tale of Personalized MedicineMilos Dokmanovic, Wen Jin Wu
Cancers|June 2, 2021
Tumor Extrinsic Factors Mediate Primary T-DM1 Resistance in HER2-Positive Breast Cancer CellsYukinori Endo, Wen Jin Wu
Nature Reviews. Cancer|January 17, 2009
Mechanism of E-cadherin lysosomal degradationWen Jin Wu, Dianne S Hirsch
Expert Review of Anticancer Therapy|February 10, 2007
Cdc42: an effector and regulator of ErbB1 as a strategic target in breast cancer therapyDianne S Hirsch, Wen Jin Wu
Cancers|July 13, 2024
Define Critical Parameters of Trastuzumab-Mediated ADCC Assays via Assay Optimization Processes, Focusing on the Impact of Cryopreserved Effector Cells on Assay PerformanceHanjing Peng, Yukinori Endo, Wen Jin Wu
Cancer Research|April 6, 2006
Growth and motility inhibition of breast cancer cells by epidermal growth factor receptor degradation is correlated with inactivation of Cdc42Dianne S Hirsch, Yi Shen, Wen Jin Wu
Cell|September 25, 2003
Activated Cdc42 sequesters c-Cbl and prevents EGF receptor degradationWen Jin Wu, Shine Tu, Richard A Cerione
The Journal of Biological Chemistry|April 13, 2005
Cdc42 and Ras cooperate to mediate cellular transformation by intersectin-LJian-Bin Wang, Wen Jin Wu, Richard A Cerione
Annual Review of Biophysics|December 21, 2021
Enzymology and Dynamics by Cryogenic Electron MicroscopyMing-Daw Tsai, Wen-Jin Wu, Meng-Chiao Ho
Pageof 9

Showing results (1-10 of 81) with videos related to

Sort By:
Pageof 9
Nature Reviews. Cardiology|September 30, 2015
Trastuzumab is not a tyrosine kinase inhibitorNishant Mohan, Wen Jin Wu
Advances in Clinical Chemistry|August 2, 2015
Monitoring Trastuzumab Resistance and Cardiotoxicity: A Tale of Personalized MedicineMilos Dokmanovic, Wen Jin Wu
Cancers|June 2, 2021
Tumor Extrinsic Factors Mediate Primary T-DM1 Resistance in HER2-Positive Breast Cancer CellsYukinori Endo, Wen Jin Wu
Nature Reviews. Cancer|January 17, 2009
Mechanism of E-cadherin lysosomal degradationWen Jin Wu, Dianne S Hirsch
Expert Review of Anticancer Therapy|February 10, 2007
Cdc42: an effector and regulator of ErbB1 as a strategic target in breast cancer therapyDianne S Hirsch, Wen Jin Wu
Cancers|July 13, 2024
Define Critical Parameters of Trastuzumab-Mediated ADCC Assays via Assay Optimization Processes, Focusing on the Impact of Cryopreserved Effector Cells on Assay PerformanceHanjing Peng, Yukinori Endo, Wen Jin Wu
Cancer Research|April 6, 2006
Growth and motility inhibition of breast cancer cells by epidermal growth factor receptor degradation is correlated with inactivation of Cdc42Dianne S Hirsch, Yi Shen, Wen Jin Wu
Cell|September 25, 2003
Activated Cdc42 sequesters c-Cbl and prevents EGF receptor degradationWen Jin Wu, Shine Tu, Richard A Cerione
The Journal of Biological Chemistry|April 13, 2005
Cdc42 and Ras cooperate to mediate cellular transformation by intersectin-LJian-Bin Wang, Wen Jin Wu, Richard A Cerione
Annual Review of Biophysics|December 21, 2021
Enzymology and Dynamics by Cryogenic Electron MicroscopyMing-Daw Tsai, Wen-Jin Wu, Meng-Chiao Ho
Pageof 9