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The American Journal of Case Reports|January 2, 2025
Cerebral Hemorrhage in Varicella-Zoster Virus Encephalitis: A Case StudyJingwen Li, Yan Lin, Liping Ni, et al.Experimental Gerontology|May 26, 2016
EGb761 improves cognitive function and regulates inflammatory responses in the APP/PS1 mouseWenbin Wan, Chunyan Zhang, Mark Danielsen, et al.Biological & Pharmaceutical Bulletin|December 3, 2020
Yi-Zhi-Fang-Dai Formula Exerts a Protective Effect on the Injury of Tight Junction Scaffold Proteins in Vitro and in Vivo by Mediating Autophagy through Regulation of the RAGE/CaMKKβ/AMPK/mTOR PathwayYuanjin Chan, Wenjing Chen, Yanjie Chen, et al.Experimental Gerontology|January 17, 2016
EGb761 protects against Aβ1-42 oligomer-induced cell damage via endoplasmic reticulum stress activation and Hsp70 protein expression increase in SH-SY5Y cellsLumei Liu, Chunyan Zhang, Bill Kalionis, et al.Journal of Neurochemistry|April 14, 2015
Aβ(1-42) oligomer-induced leakage in an in vitro blood-brain barrier model is associated with up-regulation of RAGE and metalloproteinases, and down-regulation of tight junction scaffold proteinsWenbin Wan, Lan Cao, Lumei Liu, et al.Journal of Immunology Research|August 6, 2016
An Update on Inflamm-Aging: Mechanisms, Prevention, and TreatmentShijin Xia, Xinyan Zhang, Songbai Zheng, et al.Frontiers in Neurology|February 1, 2017
Iron Deposition Leads to Neuronal α-Synuclein Pathology by Inducing Autophagy DysfunctionWenbin Wan, Lirong Jin, Zigao Wang, et al.Journal of Neuroimmunology|October 13, 2024
Effectiveness of double-filtration plasmapheresis in reducing immunoglobulin and culprit antibody levels in neuroimmune disorders: A single-center retrospective analysis from ChinaYan Lin, Xiajun Zhou, Jun Wu, et al.Frontiers in Aging Neuroscience|April 16, 2019
Decreased Function of Delayed Recall in Non-demented Elderly Subjects With Apolipoprotein E ε4 AlleleLijun Wang, Xiaoli Pan, Guoqiang Fei, et al.Neuroscience Letters|September 24, 2018
Elevated microRNA-520d-5p in the serum of patients with Parkinson's disease, possibly through regulation of cereloplasmin expressionLirong Jin, Wenbin Wan, Lingyan Wang, et al.Pageof 4