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Endocrinology|March 31, 2007
The genetics of low-density lipoprotein receptor-related protein 5 in bone: a story of extremesWendy Balemans, Wim Van HulDevelopmental Biology|October 12, 2002
Extracellular regulation of BMP signaling in vertebrates: a cocktail of modulatorsWendy Balemans, Wim Van HulBone|May 5, 2005
A generalized skeletal hyperostosis in two siblings caused by a novel mutation in the SOST geneWendy Balemans, Erna Cleiren, Ulrike Siebers, et al.Bone|June 10, 2008
Genetic analysis and effect of triiodothyronine and prednisone trial on bone turnover in a patient with craniotubular hyperostosisJosé M López, Wendy Balemans, Elke Piters, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|February 14, 2007
Novel LRP5 missense mutation in a patient with a high bone mass phenotype results in decreased DKK1-mediated inhibition of Wnt signalingWendy Balemans, Jean-Pierre Devogelaer, Erna Cleiren, et al.Nature|January 22, 2010
Essentiality of FASII pathway for Staphylococcus aureusWendy Balemans, Nacer Lounis, Ron Gilissen, et al.Calcified Tissue International|January 27, 2010
Common genetic variation in the DKK1 gene is associated with hip axis length but not with bone mineral density and bone turnover markers in young adult men: results from the Odense Androgen StudyElke Piters, Wendy Balemans, Torben Leo Nielsen, et al.Calcified Tissue International|June 4, 2008
The binding between sclerostin and LRP5 is altered by DKK1 and by high-bone mass LRP5 mutationsWendy Balemans, Elke Piters, Erna Cleiren, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|June 9, 2005
An autosomal dominant high bone mass phenotype in association with craniosynostosis in an extended family is caused by an LRP5 missense mutationMei Lan Kwee, Wendy Balemans, Erna Cleiren, et al.The Journal of Clinical Endocrinology and Metabolism|December 13, 2003
Patients with Van Buchem disease, an osteosclerotic genetic disease, have elevated bone formation markers, higher bone density, and greater derived polar moment of inertia than normalJon E Wergedal, Katarina Veskovic, Minea Hellan, et al.Pageof 2