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Wilhelm Kriz

Showing results (1-10 of 74) with videos related to

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Pediatric Nephrology (Berlin, Germany)|July 26, 2003
Progression of chronic renal failure in focal segmental glomerulosclerosis: consequence of podocyte damage or of tubulointerstitial fibrosis?Wilhelm Kriz
Microscopy Research and Technique|May 16, 2002
Podocyte is the major culprit accounting for the progression of chronic renal diseaseWilhelm Kriz
Nephron. Experimental Nephrology|June 16, 2007
Ontogenetic development of the filtration barrierWilhelm Kriz
The Journal of Clinical Investigation|September 3, 2004
Adenosine and ATP: traffic regulators in the kidneyWilhelm Kriz
Anatomical Record (Hoboken, N.J. : 2007)|October 14, 2019
The Inability of Podocytes to Proliferate: Cause, Consequences, and OriginWilhelm Kriz
Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|September 4, 2003
The pathogenesis of 'classic' focal segmental glomerulosclerosis-lessons from rat modelsWilhelm Kriz
Trends in Molecular Medicine|November 18, 2005
TRPC6 - a new podocyte gene involved in focal segmental glomerulosclerosisWilhelm Kriz
Kidney International|January 28, 2005
Pathways to nephron loss starting from glomerular diseases-insights from animal modelsWilhelm Kriz, Michel LeHir
Current Opinion in Nephrology and Hypertension|April 11, 2007
New insights into structural patterns encountered in glomerulosclerosisMichel LeHir, Wilhelm Kriz
Journal of the American Society of Nephrology : JASN|July 26, 2014
A potential role for mechanical forces in the detachment of podocytes and the progression of CKDWilhelm Kriz, Kevin V Lemley
Pageof 8

Showing results (1-10 of 74) with videos related to

Sort By:
Pageof 8
Pediatric Nephrology (Berlin, Germany)|July 26, 2003
Progression of chronic renal failure in focal segmental glomerulosclerosis: consequence of podocyte damage or of tubulointerstitial fibrosis?Wilhelm Kriz
Microscopy Research and Technique|May 16, 2002
Podocyte is the major culprit accounting for the progression of chronic renal diseaseWilhelm Kriz
Nephron. Experimental Nephrology|June 16, 2007
Ontogenetic development of the filtration barrierWilhelm Kriz
The Journal of Clinical Investigation|September 3, 2004
Adenosine and ATP: traffic regulators in the kidneyWilhelm Kriz
Anatomical Record (Hoboken, N.J. : 2007)|October 14, 2019
The Inability of Podocytes to Proliferate: Cause, Consequences, and OriginWilhelm Kriz
Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|September 4, 2003
The pathogenesis of 'classic' focal segmental glomerulosclerosis-lessons from rat modelsWilhelm Kriz
Trends in Molecular Medicine|November 18, 2005
TRPC6 - a new podocyte gene involved in focal segmental glomerulosclerosisWilhelm Kriz
Kidney International|January 28, 2005
Pathways to nephron loss starting from glomerular diseases-insights from animal modelsWilhelm Kriz, Michel LeHir
Current Opinion in Nephrology and Hypertension|April 11, 2007
New insights into structural patterns encountered in glomerulosclerosisMichel LeHir, Wilhelm Kriz
Journal of the American Society of Nephrology : JASN|July 26, 2014
A potential role for mechanical forces in the detachment of podocytes and the progression of CKDWilhelm Kriz, Kevin V Lemley
Pageof 8