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The Journal of Biological Chemistry|October 9, 2015
A Failure to Communicate: MYOSIN RESIDUES INVOLVED IN HYPERTROPHIC CARDIOMYOPATHY AFFECT INTER-DOMAIN INTERACTIONWilliam A Kronert, Girish C Melkani, Anju Melkani, et al.The Journal of Biological Chemistry|March 15, 2014
Mapping interactions between myosin relay and converter domains that power muscle functionWilliam A Kronert, Girish C Melkani, Anju Melkani, et al.Journal of Molecular Biology|January 10, 2012
Alternative relay and converter domains tune native muscle myosin isoform function in DrosophilaWilliam A Kronert, Girish C Melkani, Anju Melkani, et al.Journal of Molecular Biology|April 6, 2010
Mutating the converter-relay interface of Drosophila myosin perturbs ATPase activity, actin motility, myofibril stability and flight abilityWilliam A Kronert, Girish C Melkani, Anju Melkani, et al.Biochemical and Biophysical Research Communications|February 13, 2026
Drosophila myosin function and muscle structure are modulated by isoform-specific amino acid residues of the relay domainWilliam A Kronert, Girish C Melkani, Anju Melkani, et al.Biophysical Journal|September 4, 2004
Alternative N-terminal regions of Drosophila myosin heavy chain tune muscle kinetics for optimal power outputDouglas M Swank, William A Kronert, Sanford I Bernstein, et al.The Journal of Physiology|April 6, 2019
The R249Q hypertrophic cardiomyopathy myosin mutation decreases contractility in Drosophila by impeding force productionKaylyn M Bell, William A Kronert, Alice Huang, et al.Biophysical Journal|February 1, 2021
Prolonged myosin binding increases muscle stiffness in Drosophila models of Freeman-Sheldon syndromeKaylyn M Bell, Alice Huang, William A Kronert, et al.Journal of Molecular Biology|May 9, 2008
Alternative relay domains of Drosophila melanogaster myosin differentially affect ATPase activity, in vitro motility, myofibril structure and muscle functionWilliam A Kronert, Corey M Dambacher, Aileen F Knowles, et al.Biology|August 26, 2022
Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril StructureWilliam A Kronert, Karen H Hsu, Aditi Madan, et al.Pageof 3