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William M Dismuke

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Journal of Ocular Pharmacology and Therapeutics : the Official Journal of the Association for Ocular Pharmacology and Therapeutics|June 26, 2009
Activation of the BK(Ca) channel increases outflow facility and decreases trabecular meshwork cell volumeWilliam M Dismuke, Dorette Z Ellis
Investigative Ophthalmology & Visual Science|May 21, 2010
Endogenous regulation of human Schlemm's canal cell volume by nitric oxide signalingDorette Z Ellis, Najam A Sharif, William M Dismuke
Investigative Ophthalmology & Visual Science|December 17, 2008
Characterization of soluble guanylate cyclase in NO-induced increases in aqueous humor outflow facility and in the trabecular meshworkDorette Z Ellis, William M Dismuke, Binna M Chokshi
American Journal of Physiology. Cell Physiology|April 4, 2008
NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channelWilliam M Dismuke, Chigozirim C Mbadugha, Dorette Z Ellis
Investigative Ophthalmology & Visual Science|February 24, 2009
Human trabecular meshwork cell volume decrease by NO-independent soluble guanylate cyclase activators YC-1 and BAY-58-2667 involves the BKCa ion channelWilliam M Dismuke, Najam A Sharif, Dorette Z Ellis
Pageof 1

Showing results (1-10 of 5) with videos related to

Sort By:
Pageof 1
Journal of Ocular Pharmacology and Therapeutics : the Official Journal of the Association for Ocular Pharmacology and Therapeutics|June 26, 2009
Activation of the BK(Ca) channel increases outflow facility and decreases trabecular meshwork cell volumeWilliam M Dismuke, Dorette Z Ellis
Investigative Ophthalmology & Visual Science|May 21, 2010
Endogenous regulation of human Schlemm's canal cell volume by nitric oxide signalingDorette Z Ellis, Najam A Sharif, William M Dismuke
Investigative Ophthalmology & Visual Science|December 17, 2008
Characterization of soluble guanylate cyclase in NO-induced increases in aqueous humor outflow facility and in the trabecular meshworkDorette Z Ellis, William M Dismuke, Binna M Chokshi
American Journal of Physiology. Cell Physiology|April 4, 2008
NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channelWilliam M Dismuke, Chigozirim C Mbadugha, Dorette Z Ellis
Investigative Ophthalmology & Visual Science|February 24, 2009
Human trabecular meshwork cell volume decrease by NO-independent soluble guanylate cyclase activators YC-1 and BAY-58-2667 involves the BKCa ion channelWilliam M Dismuke, Najam A Sharif, Dorette Z Ellis
Pageof 1