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Seminars in Cell & Developmental Biology|June 12, 2020
Fated for decay: RNA elements targeted by viral endonucleasesWilliam Rodriguez, Daniel Macveigh-Fierro, Jacob Miles, et al.Viruses|September 17, 2020
Stealing the Show: KSHV Hijacks Host RNA Regulatory Pathways to Promote InfectionDaniel Macveigh-Fierro, William Rodriguez, Jacob Miles, et al.Biorxiv : the Preprint Server for Biology|June 12, 2025
IL-6 Evades KSHV-Mediated Hyperadenylation repression via CRM1-Dependent Nuclear ExportSamantha Schultz, Jacob Miles, Will Dwyer, et al.Proceedings of the National Academy of Sciences of the United States of America|February 18, 2022
The m6A reader YTHDC2 is essential for escape from KSHV SOX-induced RNA decayDaniel Macveigh-Fierro, Angelina Cicerchia, Ashley Cadorette, et al.Iscience|January 2, 2023
Nonstructural protein 1 widespread RNA decay phenotype varies among coronavirusesYahaira Bermudez, Jacob Miles, Mandy MullerViruses|June 24, 2022
Shiftless, a Critical Piece of the Innate Immune Response to Viral InfectionWilliam Rodriguez, Mandy MullerJournal of Virology|April 5, 2019
C19ORF66 Broadly Escapes Virus-Induced Endonuclease Cleavage and Restricts Kaposi's Sarcoma-Associated HerpesvirusWilliam Rodriguez, Kumaraman Srivastav, Mandy MullerBiorxiv : the Preprint Server for Biology|November 19, 2025
Virally encoded interleukin-6 (vIL-6) coordinates with human IL-6 to modulate cytokine expression during KSHV infectionYahaira Bermudez, Samantha Schultz, Jacob Miles, et al.Biorxiv : the Preprint Server for Biology|November 28, 2023
The antiviral protein Shiftless blocks p-body formation during KSHV infectionDavid Hatfield, William Rodriguez, Timothy Mehrmann, et al.The Journal of General Virology|March 13, 2026
The anti-viral protein Shiftless blocks p-body formation during KSHV infectionDavid Hatfield, William Rodriguez, Timothy Mehrmann, et al.Pageof 15