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The Journal of Biological Chemistry|August 24, 2004
alpha-synuclein is required for the fibrillar nature of ubiquitinated inclusions induced by proteasomal inhibition in primary neuronsHardy J Rideout, Paula Dietrich, Qiaohong Wang, et al.Journal of Neurochemistry|February 4, 2003
Regulation of alpha-synuclein by bFGF in cultured ventral midbrain dopaminergic neuronsHardy J Rideout, Paula Dietrich, Magali Savalle, et al.Current Topics in Developmental Biology|June 21, 2014
Mouse models of neurodevelopmental disease of the basal ganglia and associated circuitsSamuel S Pappas, Daniel K Leventhal, Roger L Albin, et al.Behavioural Brain Research|March 19, 2022
A dystonia mouse model with motor and sequencing deficits paralleling human diseaseKrista Kernodle, Allison M Bakerian, Allison Cropsey, et al.The Journal of Clinical Investigation|June 18, 2014
TorsinA hypofunction causes abnormal twisting movements and sensorimotor circuit neurodegenerationChun-Chi Liang, Lauren M Tanabe, Stephanie Jou, et al.Proceedings of the National Academy of Sciences of the United States of America|May 12, 2010
A molecular mechanism underlying the neural-specific defect in torsinA mutant miceConnie E Kim, Alex Perez, Guy Perkins, et al.Journal of Neurochemistry|September 3, 2011
Targeted disruption of neuronal 19S proteasome subunits induces the formation of ubiquitinated inclusions in the absence of cell deathAnna Droggiti, Cherry Chen-Ying Ho, Leonidas Stefanis, et al.Case Reports in Neurology|July 12, 2021
Heterozygous VPS13A and PARK2 Mutations in a Patient with Parkinsonism and SeizuresSteven D Mitchell, Roger L Albin, William T Dauer, et al.Human Molecular Genetics|November 30, 2017
TorsinA dysfunction causes persistent neuronal nuclear pore defectsSamuel S Pappas, Chun-Chi Liang, Sumin Kim, et al.The Journal of Clinical Investigation|February 2, 2021
TorsinA restoration in a mouse model identifies a critical therapeutic window for DYT1 dystoniaJay Li, Daniel S Levin, Audrey J Kim, et al.Pageof 203