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Cardiology Clinics
|
October 9, 2018
Cardiopulmonary Resuscitation Quality Issues
Essie Reed-Schrader, William T Rivers, Lynn J White, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
October 12, 2010
Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function
Jennifer A Dixon, William F Gaillard, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
September 8, 2009
Calpain inhibition preserves myocardial structure and function following myocardial infarction
Santhosh K Mani, Sundaravadivel Balasubramanian, Juozas A Zavadzkas, et al.
The Journal of Thoracic and Cardiovascular Surgery
|
March 15, 2011
Hemodynamics and myocardial blood flow patterns after placement of a cardiac passive restraint device in a model of dilated cardiomyopathy
Jennifer A Dixon, Amy M Goodman, William F Gaillard, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
August 12, 2008
Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice
Juozas A Zavadzkas, Rebecca A Plyler, Shenikqua Bouges, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
May 18, 2010
Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activity
Rupak Mukherjee, Juozas A Zavadzkas, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
June 14, 2011
Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling
Juozas A Zavadzkas, Rupak Mukherjee, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
March 22, 2015
Inhibition of class I histone deacetylase activity represses matrix metalloproteinase-2 and -9 expression and preserves LV function postmyocardial infarction
Santhosh K Mani, Christine B Kern, Denise Kimbrough, et al.
The Journal of Thoracic and Cardiovascular Surgery
|
November 8, 2011
Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature
William M Yarbrough, Rupak Mukherjee, Robert E Stroud, et al.
Journal of Cardiovascular Pharmacology
|
October 9, 2008
Aprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor I
Michel J Sabbagh, J Michael Looper, Juozas A Zavadzkas, et al.
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of 2
Search research articles
Search
Showing results (1-10 of 14) with videos related to
Sort By:
Page
of 2
Cardiology Clinics
|
October 9, 2018
Cardiopulmonary Resuscitation Quality Issues
Essie Reed-Schrader, William T Rivers, Lynn J White, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
October 12, 2010
Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function
Jennifer A Dixon, William F Gaillard, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
September 8, 2009
Calpain inhibition preserves myocardial structure and function following myocardial infarction
Santhosh K Mani, Sundaravadivel Balasubramanian, Juozas A Zavadzkas, et al.
The Journal of Thoracic and Cardiovascular Surgery
|
March 15, 2011
Hemodynamics and myocardial blood flow patterns after placement of a cardiac passive restraint device in a model of dilated cardiomyopathy
Jennifer A Dixon, Amy M Goodman, William F Gaillard, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
August 12, 2008
Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice
Juozas A Zavadzkas, Rebecca A Plyler, Shenikqua Bouges, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
May 18, 2010
Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activity
Rupak Mukherjee, Juozas A Zavadzkas, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
June 14, 2011
Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling
Juozas A Zavadzkas, Rupak Mukherjee, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
March 22, 2015
Inhibition of class I histone deacetylase activity represses matrix metalloproteinase-2 and -9 expression and preserves LV function postmyocardial infarction
Santhosh K Mani, Christine B Kern, Denise Kimbrough, et al.
The Journal of Thoracic and Cardiovascular Surgery
|
November 8, 2011
Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature
William M Yarbrough, Rupak Mukherjee, Robert E Stroud, et al.
Journal of Cardiovascular Pharmacology
|
October 9, 2008
Aprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor I
Michel J Sabbagh, J Michael Looper, Juozas A Zavadzkas, et al.
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of 2